Emanuela Argilli, Ph.D.
|Neurology||University of California, San Francisco, San Francisco, CA|
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|Madhavan A, Argilli E, Bonci A, et al. (2013) Loss of D2 dopamine receptor function modulates cocaine-induced glutamatergic synaptic potentiation in the ventral tegmental area. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 12329-36|
|Güler AD, Rainwater A, Parker JG, et al. (2012) Transient activation of specific neurons in mice by selective expression of the capsaicin receptor. Nature Communications. 3: 746|
|Ungless MA, Argilli E, Bonci A. (2010) Effects of stress and aversion on dopamine neurons: implications for addiction. Neuroscience and Biobehavioral Reviews. 35: 151-6|
|Argilli E, Sibley DR, Malenka RC, et al. (2008) Mechanism and time course of cocaine-induced long-term potentiation in the ventral tegmental area. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 9092-100|
|Zweifel LS, Argilli E, Bonci A, et al. (2008) Role of NMDA receptors in dopamine neurons for plasticity and addictive behaviors. Neuron. 59: 486-96|
|SchilstrÃ¶m B, Yaka R, Argilli E, et al. (2006) Cocaine enhances NMDA receptor-mediated currents in ventral tegmental area cells via dopamine D5 receptor-dependent redistribution of NMDA receptors. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 8549-58|
|Aicardi G, Argilli E, Cappello S, et al. (2004) Induction of long-term potentiation and depression is reflected by corresponding changes in secretion of endogenous brain-derived neurotrophic factor. Proceedings of the National Academy of Sciences of the United States of America. 101: 15788-92|