Daniel Limón

Benemerita Universidad Autónoma de Puebla (BUAP) 
Alzheimer and Parkinson models, pharmacology and neurotoxicity
"Ilhuicamina Daniel Limón Pérez de León"
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Noriega L, Díaz A, Limón D, et al. (2019) Inhibitory mechanism of 17β-aminoestrogens in the formation of Aβ aggregates. Journal of Molecular Modeling. 25: 229
Ramos-Martinez I, Martínez-Loustalot P, Lozano L, et al. (2017) Neuroinflammation induced by amyloid β25-35 modifies mucin-type O-glycosylation in the rat's hippocampus. Neuropeptides
Lozano L, Guevara J, Lefebvre T, et al. (2017) Effect of amyloid-Β (25-35) in hyperglycemic and hyperinsulinemic rats, effects on phosphorylation and O-GlcNAcylation of tau protein. Neuropeptides
Lazcano Z, Solis O, Bringas ME, et al. (2014) Unilateral injection of Aβ25-35 in the hippocampus reduces the number of dendritic spines in hyperglycemic rats. Synapse (New York, N.Y.)
Calvillo M, Diaz A, Limon DI, et al. (2013) Amyloid-β(25-35) induces a permanent phosphorylation of HSF-1, but a transitory and inflammation-independent overexpression of Hsp-70 in C6 astrocytoma cells. Neuropeptides. 47: 339-46
Diaz A, Limon D, Chávez R, et al. (2012) Aβ25-35 injection into the temporal cortex induces chronic inflammation that contributes to neurodegeneration and spatial memory impairment in rats. Journal of Alzheimer's Disease : Jad. 30: 505-22
Limón D, Díaz A, Hernandez M, et al. (2012) Corrigendum to “Neuroprotective effect of the aminoestrogen prolame against impairment of learning and memory skills in rats injected with amyloid-β-25–35 into the hippocampus” [Eur. J. Pharmacol. 685 (2012) 74–80] European Journal of Pharmacology. 689: 294
Zomosa-Signoret V, Mayoral M, Limón D, et al. (2011) Sialylated and O-glycosidically linked glycans in prion protein deposits in a case of Gerstmann-Sträussler-Scheinker disease. Neuropathology : Official Journal of the Japanese Society of Neuropathology. 31: 162-9
Garcia E, Limon D, Perez-De La Cruz V, et al. (2008) Lipid peroxidation, mitochondrial dysfunction and neurochemical and behavioural deficits in different neurotoxic models: protective role of S-allylcysteine. Free Radical Research. 42: 892-902
Pérez-Severiano F, Salvatierra-Sánchez R, Rodríguez-Pérez M, et al. (2004) S-Allylcysteine prevents amyloid-beta peptide-induced oxidative stress in rat hippocampus and ameliorates learning deficits. European Journal of Pharmacology. 489: 197-202
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