Jürgen Wess
Affiliations: | NIDDK, National Institutes of Health |
Google:
"Jürgen Wess"Mean distance: (not calculated yet)
Collaborators
Sign in to add collaboratorMorgane Thomsen | collaborator | 2002- | NIDDK, National Institutes of Health |
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Balla H, Borsodi K, Őrsy P, et al. (2023) Intracellular signaling pathways of muscarinic acetylcholine receptor-mediated detrusor muscle contractions. American Journal of Physiology. Renal Physiology |
Zheng F, Wess J, Alzheimer C. (2023) Long-Term-But Not Short-Term-Plasticity at the Mossy Fiber-CA3 Pyramidal Cell Synapse in Hippocampus Is Altered in M1/M3 Muscarinic Acetylcholine Receptor Double Knockout Mice. Cells. 12 |
Wess J. (2023) The third intracellular loop of GPCRs: size matters. Trends in Pharmacological Sciences |
Meister J, Bone DBJ, Knudsen JR, et al. (2022) Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells. Nature Communications. 13: 22 |
Wess J. (2021) In Vivo Metabolic Roles of G Proteins of the Gi Family Studied with Novel Mouse Models. Endocrinology |
Wang L, Zhu L, Meister J, et al. (2020) Use of DREADD Technology to Identify Novel Targets for Antidiabetic Drugs. Annual Review of Pharmacology and Toxicology |
Pydi SP, Jain S, Barella LF, et al. (2020) β-arrestin-1 suppresses myogenic reprogramming of brown fat to maintain euglycemia. Science Advances. 6: eaba1733 |
Wang L, Pydi SP, Zhu L, et al. (2020) Adipocyte G signaling is essential for maintaining whole-body glucose homeostasis and insulin sensitivity. Nature Communications. 11: 2995 |
Zhu L, Rossi M, Doliba NM, et al. (2020) Beta-cell M muscarinic acetylcholine receptors as potential targets for novel antidiabetic drugs. International Immunopharmacology. 81: 106267 |
Dattaroy D, Barella LF, Pydi SP, et al. (2020) 2129-P: Glucagon Secretion from Pancreatic Islets Is Regulated by Beta-Arrestin-1 Diabetes. 69 |