Hartmut Cuny
Affiliations: | 2009-2015 | RMIT University, Melbourne, Victoria, Australia |
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| Cuny H, Yu R, Tae HS, et al. (2017) α-Conotoxins active at α3-containing nicotinic acetylcholine receptors and their molecular determinants for selective inhibition. British Journal of Pharmacology |
| Cuny H, Kompella SN, Tae HS, et al. (2016) Key Structural Determinants in the Agonist Binding Loops of Human β2 and β4 Nicotinic Acetylcholine Receptor Subunits Contribute to α3β4 Subtype Selectivity of α-Conotoxins. The Journal of Biological Chemistry |
| Kompella SN, Cuny H, Hung A, et al. (2015) Molecular Basis for Differential Sensitivity of α-Conotoxin RegIIA at Rat and Human Neuronal Nicotinic Acetylcholine Receptors. Molecular Pharmacology. 88: 993-1001 |
| Huynh TG, Cuny H, Slesinger PA, et al. (2015) Novel mechanism of voltage-gated N-type (Cav2.2) calcium channel inhibition revealed through α-conotoxin Vc1.1 activation of the GABA(B) receptor. Molecular Pharmacology. 87: 240-50 |
| Berecki G, McArthur JR, Cuny H, et al. (2014) Differential Cav2.1 and Cav2.3 channel inhibition by baclofen and α-conotoxin Vc1.1 via GABAB receptor activation. The Journal of General Physiology. 143: 465-79 |
| Grishin AA, Cuny H, Hung A, et al. (2013) Identifying key amino acid residues that affect α-conotoxin AuIB inhibition of α3β4 nicotinic acetylcholine receptors. The Journal of Biological Chemistry. 288: 34428-42 |
| Inserra MC, Kompella SN, Vetter I, et al. (2013) Isolation and characterization of α-conotoxin LsIA with potent activity at nicotinic acetylcholine receptors. Biochemical Pharmacology. 86: 791-9 |
| Yasuda T, Cuny H, Adams DJ. (2013) Kv3.1 channels stimulate adult neural precursor cell proliferation and neuronal differentiation. The Journal of Physiology. 591: 2579-91 |
| Adams D, Harper A, Grishin A, et al. (2013) Actions of α-conotoxins on neuronal nicotinic acetylcholine receptors and synaptic transmission in rat parasympathetic ganglia Autonomic Neuroscience. 177: 306 |
| Cuny H, de Faoite A, Huynh TG, et al. (2012) γ-Aminobutyric acid type B (GABAB) receptor expression is needed for inhibition of N-type (Cav2.2) calcium channels by analgesic α-conotoxins. The Journal of Biological Chemistry. 287: 23948-57 |