Lingjie He
Affiliations: | 2008-2015 | Institute of Neuroscence, CAS, Eugene, OR, United States |
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Parents
Sign in to add mentorXiaohui Zhang | grad student | 2008-2015 | Institute of Neuroscence, CAS |
Huda Y. Zoghbi | post-doc | 2016- | Baylor College of Medicine |
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Publications
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He L, Caudill MS, Jing J, et al. (2022) A weakened recurrent circuit in the hippocampus of Rett syndrome mice disrupts long-term memory representations. Neuron |
Achilly NP, He LJ, Kim OA, et al. (2021) Deleting from the cerebellum rather than its neuronal subtypes causes a delay in motor learning in mice. Elife. 10 |
Yu B, Yuan B, Dai JK, et al. (2020) Reversal of Social Recognition Deficit in Adult Mice with MECP2 Duplication via Normalization of MeCP2 in the Medial Prefrontal Cortex. Neuroscience Bulletin |
Tong DL, Chen RG, Lu YL, et al. (2019) The critical role of ASD-related gene CNTNAP3 in regulating synaptic development and social behavior in mice. Neurobiology of Disease. 104486 |
Wang L, Pang K, Han K, et al. (2019) An autism-linked missense mutation in SHANK3 reveals the modularity of Shank3 function. Molecular Psychiatry |
Lu H, Ash RT, He L, et al. (2016) Loss and Gain of MeCP2 Cause Similar Hippocampal Circuit Dysfunction that Is Rescued by Deep Brain Stimulation in a Rett Syndrome Mouse Model. Neuron |
Meng X, Wang W, Lu H, et al. (2016) Manipulations of MeCP2 in glutamatergic neurons highlight their contributions to Rett and other neurological disorders. Elife. 5 |
Ure K, Lu H, Wang W, et al. (2016) Restoration of Mecp2 expression in GABAergic neurons is sufficient to rescue multiple disease features in a mouse model of Rett Syndrome. Elife. 5 |
Meng X, Wang W, Lu H, et al. (2016) Author response: Manipulations of MeCP2 in glutamatergic neurons highlight their contributions to Rett and other neurological disorders Elife |
Ure K, Lu H, Wang W, et al. (2016) Author response: Restoration of Mecp2 expression in GABAergic neurons is sufficient to rescue multiple disease features in a mouse model of Rett syndrome Elife |