Yutaka Yoshida

Affiliations: 
Cincinnati Children's Hospital Medical Center, Cincinnati, OH, United States 
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Publications

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Ueno M, Nakamura Y, Nakagawa H, et al. (2020) Olig2-Induced Semaphorin Expression Drives Corticospinal Axon Retraction After Spinal Cord Injury. Cerebral Cortex (New York, N.Y. : 1991)
Gu Z, Koppel N, Kalamboglas J, et al. (2020) Semaphorin-mediated corticospinal axon elimination depends on the activity-induced Bax/Bak-caspase pathway. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Gu Z, Ueno M, Klinefelter K, et al. (2019) Skilled movements in mice require inhibition of corticospinal axon collateral formation in the spinal cord by semaphorin signaling. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Ueno M, Nakamura Y, Li J, et al. (2018) Corticospinal Circuits from the Sensory and Motor Cortices Differentially Regulate Skilled Movements through Distinct Spinal Interneurons. Cell Reports. 23: 1286-1300.e7
Imai F, Yoshida Y. (2017) Molecular mechanisms underlying monosynaptic sensory-motor circuit development in the spinal cord. Developmental Dynamics : An Official Publication of the American Association of Anatomists
Gu Z, Kalamboglas J, Yoshioka S, et al. (2017) Control of species-dependent cortico-motoneuronal connections underlying manual dexterity. Science (New York, N.Y.). 357: 400-404
Gu Z, Serradj N, Ueno M, et al. (2017) Skilled Movements Require Non-apoptotic Bax/Bak Pathway-Mediated Corticospinal Circuit Reorganization. Neuron. 94: 626-641.e4
Imai F, Chen X, Weirauch MT, et al. (2016) Requirement for Dicer in Maintenance of Monosynaptic Sensory-Motor Circuits in the Spinal Cord. Cell Reports. 17: 2163-2172
Imai F, Ladle DR, Leslie JR, et al. (2016) Synapse Formation in Monosynaptic Sensory-Motor Connections Is Regulated by Presynaptic Rho GTPase Cdc42. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 5724-35
Wehner AB, Abdesselem H, Dickendesher TL, et al. (2016) Semaphorin 3A is a retrograde cell death signal in developing sympathetic neurons. Development (Cambridge, England). 143: 1560-70
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