Carlos Cruchaga

Washington University School of Medicine, St. Louis, MO, United States 
Alzheimer's, genetics, genomics, quantitative traits, WES, WGS, proteomics
"Carlos Cruchaga"
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Dammer EB, Shantaraman A, Ping L, et al. (2023) Proteomic Network Analysis of Alzheimer's Disease Cerebrospinal Fluid Reveals Alterations Associated with ε4 Genotype and Atomoxetine Treatment. Medrxiv : the Preprint Server For Health Sciences
Gorijala P, Aslam MM, Dang LT, et al. (2023) Alzheimer's polygenic risk scores are associated with cognitive phenotypes in Down syndrome. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
Shirzadi Z, Schultz SA, Yau WW, et al. (2023) Etiology of White Matter Hyperintensities in Autosomal Dominant and Sporadic Alzheimer Disease. Jama Neurology
Neumann A, Ohlei O, Küçükali F, et al. (2023) Multivariate GWAS of Alzheimer's disease CSF biomarker profiles implies GRIN2D in synaptic functioning. Genome Medicine. 15: 79
Wagemann O, Li Y, Hassenstab J, et al. (2023) Investigation of sex differences in mutation carriers of the Dominantly Inherited Alzheimer Network. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
Johnson ECB, Bian S, Haque RU, et al. (2023) Cerebrospinal fluid proteomics define the natural history of autosomal dominant Alzheimer's disease. Nature Medicine
Dawson T, Rentia U, Sanford J, et al. (2023) Locus specific endogenous retroviral expression associated with Alzheimer's disease. Frontiers in Aging Neuroscience. 15: 1186470
Rajabli F, Benchek P, Tosto G, et al. (2023) Multi-ancestry genome-wide meta-analysis of 56,241 individuals identifies and nominates ancestry-specific loci , , and as novel risk loci for Alzheimer's disease: the Alzheimer's Disease Genetics Consortium. Medrxiv : the Preprint Server For Health Sciences
McKay NS, Gordon BA, Hornbeck RC, et al. (2023) Positron emission tomography and magnetic resonance imaging methods and datasets within the Dominantly Inherited Alzheimer Network (DIAN). Nature Neuroscience
Phillips B, Western D, Wang L, et al. (2023) Proteome wide association studies of LRRK2 variants identify novel causal and druggable proteins for Parkinson's disease. Npj Parkinson's Disease. 9: 107
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