Yanlin He

Affiliations: 
2014-2019 Children's Nutrition Research Center Baylor College of Medicine, Houston, TX 
 2019- Brain Glycemic and Metabolism Control Pennington Biomedical Research Center 
Area:
diabetes, obesity, depression, ion channel, PD
Google:
"Yanlin He"
Mean distance: (not calculated yet)
 
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Tu L, He Y, Xu Y. (2023) Anoctamin 4 defines glucose-inhibited neurons in the ventromedial hypothalamus. Neural Regeneration Research. 19: 1177-1178
Feng B, Harms J, Chen E, et al. (2023) Current Discoveries and Future Implications of Eating Disorders. International Journal of Environmental Research and Public Health. 20
Tu L, Bean JC, He Y, et al. (2023) Anoctamin 4 channel currents activate glucose-inhibited neurons in the mouse ventromedial hypothalamus during hypoglycemia. The Journal of Clinical Investigation
Feng B, Liu H, Mishra I, et al. (2023) Asprosin promotes feeding through SK channel-dependent activation of AgRP neurons. Science Advances. 9: eabq6718
Yu M, Bean JC, Liu H, et al. (2022) SK3 in POMC neurons plays a sexually dimorphic role in energy and glucose homeostasis. Cell & Bioscience. 12: 170
Pei Z, He Y, Bean JC, et al. (2022) Gabra5 plays a sexually dimorphic role in POMC neuron activity and glucose balance. Frontiers in Endocrinology. 13: 889122
Torres Irizarry VC, Jiang Y, He Y, et al. (2022) Hypothalamic Estrogen Signaling and Adipose Tissue Metabolism in Energy Homeostasis. Frontiers in Endocrinology. 13: 898139
Cai X, Liu H, Feng B, et al. (2022) Publisher Correction: A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice. Nature Neuroscience
Cai X, Liu H, Feng B, et al. (2022) A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice. Nature Neuroscience. 25: 646-658
Mishra I, Xie WR, Bournat JC, et al. (2022) Protein tyrosine phosphatase receptor δ serves as the orexigenic asprosin receptor. Cell Metabolism
See more...