Alan Faden, M.D.
Affiliations: | Georgetown University, Washington, DC |
Area:
traumatic brain injury, spinal cord injuryGoogle:
"Alan Faden"Mean distance: 20.25 (cluster 29)
Cross-listing: Neurotrauma Tree
Children
Sign in to add trainee| Jason W. Allen | grad student | 2000 | Georgetown University Medical Center (Neurotrauma Tree) |
| Jacob Skovira | grad student | 2014 | University of Maryland Medical School (Neurotrauma Tree) |
| Benedict C Albensi | post-doc | Georgetown | |
| Kimberly R. Byrnes | post-doc | Uniformed Services University | |
| Shruti V. Kabadi | post-doc | ||
| David Loane | post-doc | Georgetown | |
| Ahdeah Pajoohesh-Ganji | post-doc | (E-Tree) |
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Publications
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| Ritzel RM, Li Y, Jiao Y, et al. (2023) The brain-bone marrow axis and its implications for chronic traumatic brain injury. Research Square |
| Li Y, Khan N, Ritzel RM, et al. (2023) Sexually dimorphic extracellular vesicle responses after chronic spinal cord injury are associated with neuroinflammation and neurodegeneration in the aged brain. Journal of Neuroinflammation. 20: 197 |
| Henry RJ, Barrett JP, Vaida M, et al. (2023) Interaction of high-fat diet and brain trauma alters adipose tissue macrophages and brain microglia associated with exacerbated cognitive dysfunction. Biorxiv : the Preprint Server For Biology |
| Lei Z, Krishnamachary B, Ritzel RM, et al. (2023) Age-related changes in plasma extracellular vesicles influence neuroinflammation in the brain and neurological outcome after traumatic spinal cord injury. Research Square |
| Ritzel RM, Li Y, Jiao Y, et al. (2023) Brain injury accelerates the onset of a reversible age-related microglial phenotype associated with inflammatory neurodegeneration. Science Advances. 9: eadd1101 |
| Ritzel RM, Li Y, Lei Z, et al. (2022) Functional and transcriptional profiling of microglial activation during the chronic phase of TBI identifies an age-related driver of poor outcome in old mice. Geroscience |
| Li Y, Ritzel RM, Lei Z, et al. (2021) Sexual dimorphism in neurological function after SCI is associated with disrupted neuroinflammation in both injured spinal cord and brain. Brain, Behavior, and Immunity. 101: 1-22 |
| Faden AI, Barrett JP, Stoica BA, et al. (2021) Bidirectional Brain-Systemic Interactions and Outcomes After TBI. Trends in Neurosciences |
| Hanscom M, Loane DJ, Aubretch T, et al. (2021) Acute colitis during chronic experimental traumatic brain injury in mice induces dysautonomia and persistent extraintestinal, systemic, and CNS inflammation with exacerbated neurological deficits. Journal of Neuroinflammation. 18: 24 |
| Khan N, Cao T, He J, et al. (2020) Spinal cord injury alters microRNA and CD81+ exosome levels in plasma extracellular nanoparticles with neuroinflammatory potential. Brain, Behavior, and Immunity |