S. Rock Levinson
Affiliations: | 1970-1975 | Physiological Laboratory | University of Cambridge, Cambridge, England, United Kingdom |
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| Wang ZJ, Tabakoff B, Levinson SR, et al. (2015) Inhibition of Nav1.7 channels by methyl eugenol as a mechanism underlying its antinociceptive and anesthetic actions. Acta Pharmacologica Sinica. 36: 791-9 |
| Frenz CT, Hansen A, Dupuis ND, et al. (2014) NaV1.5 sodium channel window currents contribute to spontaneous firing in olfactory sensory neurons. Journal of Neurophysiology. 112: 1091-104 |
| Wang ZJ, Levinson SR, Sun L, et al. (2014) Identification of both GABAA receptors and voltage-activated Na(+) channels as molecular targets of anticonvulsant α-asarone. Frontiers in Pharmacology. 5: 40 |
| Gould HJ, England JD, Soignier RD, et al. (2004) Ibuprofen blocks changes in Na v 1.7 and 1.8 sodium channels associated with complete Freund's adjuvant-induced inflammation in rat. The Journal of Pain : Official Journal of the American Pain Society. 5: 270-80 |
| Caldwell JH, Levinson SR. (2004) The biology of voltage-gatedsodium channels Advances in Molecular and Cell Biology. 32: 15-50 |
| Wang ZJ, Snell LD, Tabakoff B, et al. (2002) Inhibition of neuronal Na+ channels by the novel antiepileptic compound DCUKA: identification of the diphenylureido moiety as an inactivation modifier. Experimental Neurology. 178: 129-38 |
| Shcherbatko AD, Davenport CM, Speh JC, et al. (2001) Progesterone treatment abolishes exogenously expressed ionic currents in Xenopus oocytes. American Journal of Physiology. Cell Physiology. 280: C677-88 |
| Vabnick I, Trimmer JS, Schwarz TL, et al. (1999) Dynamic potassium channel distributions during axonal development prevent aberrant firing patterns. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 19: 747-58 |
| Novakovic SD, Levinson SR, Schachner M, et al. (1998) Disruption and reorganization of sodium channels in experimental allergic neuritis. Muscle & Nerve. 21: 1019-32 |
| Bennett E, Urcan MS, Tinkle SS, et al. (1997) Contribution of sialic acid to the voltage dependence of sodium channel gating. A possible electrostatic mechanism. The Journal of General Physiology. 109: 327-43 |