Lakshya Bajaj, Ph.D.
Affiliations: | 2012-2018 | Molecular and Human Genetics | Baylor College of Medicine, Houston, TX |
| 2019- | Genetics | Harvard Medical School, Boston, MA, United States |
Area:
Genetics, Cell and molecular biology, Epigenetics, BiochemistryGoogle:
"Lakshya Bajaj"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Marco Sardiello | grad student | 2012-2018 | Baylor |
| Scott G. Kennedy | post-doc | 2019- | Harvard Medical School |
| Victoria P. Belancio | research scientist | 2011-2012 | Tulane Medical School (Chemistry Tree) |
Collaborators
Sign in to add collaborator| Randy W. Schekman | collaborator | 2016-2018 | UC Berkeley (Cell Biology Tree) | |
| (Vesicle Budding Assay) | ||||
| Rui Chen | collaborator | 2017-2018 | Baylor | |
| (Electroretinography) | ||||
BETA: Related publications
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Publications
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| Bajaj L, Sharma J, di Ronza A, et al. (2020) A CLN6-CLN8 complex recruits lysosomal enzymes at the ER for Golgi transfer. The Journal of Clinical Investigation |
| di Ronza A, Bajaj L, Sharma J, et al. (2018) CLN8 is an endoplasmic reticulum cargo receptor that regulates lysosome biogenesis. Nature Cell Biology |
| Martini-Stoica H, Cole AL, Swartzlander DB, et al. (2018) TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. Journal of Experimental Medicine |
| Martini-Stoica H, Cole AL, Swartzlander DB, et al. (2018) TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. The Journal of Experimental Medicine |
| Bajaj L, Lotfi P, Pal R, et al. (2018) Lysosome biogenesis in health and disease. Journal of Neurochemistry |
| Bajaj L, Lotfi P, Pal R, et al. (2018) Lysosome biogenesis in health and disease. Journal of Neurochemistry |
| Palmieri M, Pal R, Nelvagal HR, et al. (2017) Corrigendum: mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications. 8: 15793 |
| Palmieri M, Pal R, Nelvagal HR, et al. (2017) mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications |
| Palmieri M, Pal R, Nelvagal HR, et al. (2017) mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications. 8: 14338 |
| Pal R, Bajaj L, Sharma J, et al. (2016) NADPH oxidase promotes Parkinsonian phenotypes by impairing autophagic flux in an mTORC1-independent fashion in a cellular model of Parkinson's disease. Scientific Reports |