Lakshya Bajaj, Ph.D.

Affiliations: 
2012-2018 Molecular and Human Genetics Baylor College of Medicine, Houston, TX 
 2019- Genetics Harvard Medical School, Boston, MA, United States 
Area:
Genetics, Cell and molecular biology, Epigenetics, Biochemistry
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"Lakshya Bajaj"
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Parents

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Marco Sardiello grad student 2012-2018 Baylor
Scott G. Kennedy post-doc 2019- Harvard Medical School
Victoria P. Belancio research scientist 2011-2012 Tulane Medical School (Chemistry Tree)

Collaborators

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Randy W. Schekman collaborator 2016-2018 UC Berkeley (Cell Biology Tree)
 (Vesicle Budding Assay)
Rui Chen collaborator 2017-2018 Baylor
 (Electroretinography)
BETA: Related publications

Publications

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Bajaj L, Sharma J, di Ronza A, et al. (2020) A CLN6-CLN8 complex recruits lysosomal enzymes at the ER for Golgi transfer. The Journal of Clinical Investigation
di Ronza A, Bajaj L, Sharma J, et al. (2018) CLN8 is an endoplasmic reticulum cargo receptor that regulates lysosome biogenesis. Nature Cell Biology
Martini-Stoica H, Cole AL, Swartzlander DB, et al. (2018) TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. Journal of Experimental Medicine
Martini-Stoica H, Cole AL, Swartzlander DB, et al. (2018) TFEB enhances astroglial uptake of extracellular tau species and reduces tau spreading. The Journal of Experimental Medicine
Bajaj L, Lotfi P, Pal R, et al. (2018) Lysosome biogenesis in health and disease. Journal of Neurochemistry
Bajaj L, Lotfi P, Pal R, et al. (2018) Lysosome biogenesis in health and disease. Journal of Neurochemistry
Palmieri M, Pal R, Nelvagal HR, et al. (2017) Corrigendum: mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications. 8: 15793
Palmieri M, Pal R, Nelvagal HR, et al. (2017) mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications
Palmieri M, Pal R, Nelvagal HR, et al. (2017) mTORC1-independent TFEB activation via Akt inhibition promotes cellular clearance in neurodegenerative storage diseases. Nature Communications. 8: 14338
Pal R, Bajaj L, Sharma J, et al. (2016) NADPH oxidase promotes Parkinsonian phenotypes by impairing autophagic flux in an mTORC1-independent fashion in a cellular model of Parkinson's disease. Scientific Reports
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