Bradley K. Taylor

Anesthesiology and Perioperative Medicine University of Pittsburgh, Pittsburgh, PA, United States 
"Bradley Taylor"
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Cooper AH, Hedden NS, Corder G, et al. (2021) Endogenous µ-opioid receptor activity in the lateral and capsular subdivisions of the right central nucleus of the amygdala prevents chronic postoperative pain. Journal of Neuroscience Research
Cooper AH, Brightwell JJ, Hedden NS, et al. (2018) The left central nucleus of the amygdala contributes to mechanical allodynia and hyperalgesia following right-sided peripheral nerve injury. Neuroscience Letters
Kaushal R, Taylor BK, Jamal AB, et al. (2016) GABA-A receptor activity in the noradrenergic locus coeruleus drives trigeminal neuropathic pain in the rat; contribution of NAα1 receptors in the medial prefrontal cortex. Neuroscience
Rau KK, Hill CE, Harrison BJ, et al. (2016) Cutaneous tissue damage induces long-lasting nociceptive sensitization and regulation of cellular stress- and nerve injury-associated genes in sensory neurons. Experimental Neurology
Griggs RB, Donahue RR, Adkins BG, et al. (2015) Pioglitazone inhibits the development of hyperalgesia and sensitization of spinal nociresponsive neurons in type 2 diabetes. The Journal of Pain : Official Journal of the American Pain Society
Griggs RB, Bardo MT, Taylor BK. (2015) Gabapentin alleviates affective pain after traumatic nerve injury. Neuroreport. 26: 522-7
Fehrenbacher JC, Loverme J, Clarke W, et al. (2009) Rapid pain modulation with nuclear receptor ligands. Brain Research Reviews. 60: 114-24
Guo N, Garcia MM, Taylor BK, et al. (2008) Blockade of micro-opioid receptors in the medial thalamus inhibits acquisition, but not expression, of morphine-induced conditioned place preference. Neuroscience. 151: 948-54
Taylor BK, Abhyankar SS, Vo NT, et al. (2007) Neuropeptide Y acts at Y1 receptors in the rostral ventral medulla to inhibit neuropathic pain. Pain. 131: 83-95
Bivalacqua TJ, Deng W, Kendirci M, et al. (2007) Mesenchymal stem cells alone or ex vivo gene modified with endothelial nitric oxide synthase reverse age-associated erectile dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. 292: H1278-90
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