Sruti Rayaprolu, Ph.D.

Affiliations: 
Neurology Emory University School of Medicine, Atlanta, GA, United States 
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"Sruti Rayaprolu"
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Publications

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Wynne ME, Ogunbona O, Lane AR, et al. (2023) APOE expression and secretion are modulated by mitochondrial dysfunction. Elife. 12
Rayaprolu S, Bitarafan S, Santiago JV, et al. (2022) Cell type-specific biotin labeling in vivo resolves regional neuronal and astrocyte proteomic differences in mouse brain. Nature Communications. 13: 2927
Ramesha S, Rayaprolu S, Bowen CA, et al. (2021) Unique molecular characteristics and microglial origin of Kv1.3 channel-positive brain myeloid cells in Alzheimer's disease. Proceedings of the National Academy of Sciences of the United States of America. 118
Rayaprolu S, Higginbotham L, Bagchi P, et al. (2020) Systems-based proteomics to resolve the biology of Alzheimer's disease beyond amyloid and tau. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Rayaprolu S, Gao T, Xiao H, et al. (2020) Flow-cytometric microglial sorting coupled with quantitative proteomics identifies moesin as a highly-abundant microglial protein with relevance to Alzheimer's disease. Molecular Neurodegeneration. 15: 28
Moloney C, Rayaprolu S, Howard J, et al. (2018) Analysis of spinal and muscle pathology in transgenic mice overexpressing wild-type and ALS-linked mutant MATR3. Acta Neuropathologica Communications. 6: 137
Rayaprolu S, Seven YB, Howard J, et al. (2018) Partial loss of ATP13A2 causes selective gliosis independent of robust lipofuscinosis. Molecular and Cellular Neurosciences
Moloney C, Rayaprolu S, Howard J, et al. (2017) Retraction Note: Transgenic mice overexpressing the ALS-linked protein Matrin 3 develop a profound muscle phenotype. Acta Neuropathologica Communications. 5: 97
Khare S, Nick JA, Zhang Y, et al. (2017) A KCNC3 mutation causes a neurodevelopmental, non-progressive SCA13 subtype associated with dominant negative effects and aberrant EGFR trafficking. Plos One. 12: e0173565
Moloney C, Rayaprolu S, Howard J, et al. (2016) Transgenic mice overexpressing the ALS-linked protein Matrin 3 develop a profound muscle phenotype. Acta Neuropathologica Communications. 4: 122
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