Ana Jovicic
Affiliations: | Genentech, Inc., San Francisco, CA, United States |
Area:
neurodegeneration, stem cellsGoogle:
"Ana Jovicic"Mean distance: (not calculated yet)
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Publications
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| Kramer NJ, Haney MS, Morgens DW, et al. (2018) CRISPR-Cas9 screens in human cells and primary neurons identify modifiers of C9ORF72 dipeptide-repeat-protein toxicity. Nature Genetics |
| Jovičić A, Gitler AD. (2017) Distinct repertoires of microRNAs present in mouse astrocytes compared to astrocyte-secreted exosomes. Plos One. 12: e0171418 |
| Dikiy I, Fauvet B, Jovičić A, et al. (2016) Semisynthetic and in vitro phosphorylation of alpha-synuclein at Y39 promotes functional partly-helical membrane-bound states resembling those induced by PD mutations. Acs Chemical Biology |
| Lee S, Shang Y, Redmond SA, et al. (2016) Activation of HIPK2 Promotes ER Stress-Mediated Neurodegeneration in Amyotrophic Lateral Sclerosis. Neuron |
| Jovičić A, Paul JW, Gitler AD. (2016) Nuclear transport dysfunction: a common theme in amyotrophic lateral sclerosis and frontotemporal dementia. Journal of Neurochemistry |
| Boeynaems S, Bogaert E, Michiels E, et al. (2016) Drosophila screen connects nuclear transport genes to DPR pathology in c9ALS/FTD. Scientific Reports. 6: 20877 |
| Jovičić A, Mertens J, Boeynaems S, et al. (2015) Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALS. Nature Neuroscience. 18: 1226-9 |
| Jovičič A, Mertens J, Boeynaems S, et al. (2015) Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALS Nature Neuroscience. 18: 1226-1229 |