William M. Meil, PhD
Affiliations: | Psychology | IUP |
Area:
neuroscience, addiction, drugs and behaviorWebsite:
http://www.iup.edu/page.aspx?id=44397Google:
"William Meil"Mean distance: 15.91 (cluster 19) | S | N | B | C | P |
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Publications
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Kozikowski AP, Araldi GL, Boja J, et al. (1998) Chemistry and pharmacology of the piperidine-based analogues of cocaine. Identification of potent DAT inhibitors lacking the tropane skeleton. Journal of Medicinal Chemistry. 41: 1962-9 |
Meil WM, Schechter MD. (1997) Olanzapine attenuates the reinforcing effects of cocaine. European Journal of Pharmacology. 340: 17-26 |
Pierce RC, Meil WM, Kalivas PW. (1997) The NMDA antagonist, dizocilpine, enhances cocaine reinforcement without influencing mesoaccumbens dopamine transmission. Psychopharmacology. 133: 188-95 |
Meil WM, See RE. (1997) Lesions of the basolateral amygdala abolish the ability of drug associated cues to reinstate responding during withdrawal from self-administered cocaine. Behavioural Brain Research. 87: 139-48 |
Roll JM, McSweeney FK, Meil WM, et al. (1996) A preliminary examination of some effects of cocaine on within-session patterns of responding. Behavioural Processes. 37: 9-20 |
Meil WM, See RE. (1996) Conditioned cued recovery of responding following prolonged withdrawal from self-administered cocaine in rats: an animal model of relapse. Behavioural Pharmacology. 7: 754-763 |
Meil WM, Roll JM, Grimm JW, et al. (1995) Tolerance-like attenuation to contingent and noncontingent cocaine-induced elevation of extracellular dopamine in the ventral striatum following 7 days of withdrawal from chronic treatment. Psychopharmacology. 118: 338-46 |
Meil W, See RE. (1994) Single preexposure to fluphenazine produces persisting behavioral sensitization accompanied by tolerance to fluphenazine-induced striatal dopamine overflow in rats. Pharmacology, Biochemistry, and Behavior. 48: 605-12 |