Alicia L. Hawthorne, Ph.D.
Affiliations: | Cell Biology | Emory University, Atlanta, GA |
Area:
Neuroscience, Spinal cord injuryGoogle:
"Alicia Hawthorne"Mean distance: 16.53 (cluster 11) | S | N | B | C | P |
Parents
Sign in to add mentor| Jerry Silver | grad student | 2003-2011 | Case Western |
| Gary J. Bassell | post-doc | Emory |
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Publications
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| Kalinski AL, Kar AN, Craver J, et al. (2019) Deacetylation of Miro1 by HDAC6 blocks mitochondrial transport and mediates axon growth inhibition. The Journal of Cell Biology |
| Urbanska AS, Janusz-Kaminska A, Switon K, et al. (2017) ZBP1 phosphorylation at serine 181 regulates its dendritic transport and the development of dendritic trees of hippocampal neurons. Scientific Reports. 7: 1876 |
| Hawthorne AL. (2014) Repurposing Reelin: the new role of radial glia, Reelin and Notch in motor neuron migration. Experimental Neurology. 256: 17-20 |
| Hawthorne AL, Hu H, Kundu B, et al. (2011) The unusual response of serotonergic neurons after CNS Injury: lack of axonal dieback and enhanced sprouting within the inhibitory environment of the glial scar. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 5605-16 |
| Hawthorne AL, Popovich PG. (2011) Emerging concepts in myeloid cell biology after spinal cord injury. Neurotherapeutics : the Journal of the American Society For Experimental Neurotherapeutics. 8: 252-61 |
| Hawthorne AL, Wylie CJ, Landmesser LT, et al. (2010) Serotonergic neurons migrate radially through the neuroepithelium by dynamin-mediated somal translocation. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 420-30 |
| Busch SA, Horn KP, Cuascut FX, et al. (2010) Adult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 255-65 |
| Horn KP, Busch SA, Hawthorne AL, et al. (2008) Another barrier to regeneration in the CNS: activated macrophages induce extensive retraction of dystrophic axons through direct physical interactions. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 9330-41 |