Emily Workman
Affiliations: | Neuroscience | University of Texas at Austin, Austin, Texas, U.S.A. |
Area:
depression, mTORGoogle:
"Emily Workman"Mean distance: 15.58 (cluster 11) | S | N | B | C | P |
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Publications
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| Workman ER, Niere F, Raab-Graham KF. (2017) Engaging homeostatic plasticity to treat depression. Molecular Psychiatry. 23: 26-35 |
| Heaney CF, Workman ER, Ericsson EK, et al. (2017) Rapid antidepressant synapse formation requires GABAB receptor-activation of mTORC1 Alcohol. 60: 235 |
| Wolfe SA, Workman ER, Heaney CF, et al. (2016) FMRP regulates an ethanol-dependent shift in GABABR function and expression with rapid antidepressant properties. Nature Communications. 7: 12867 |
| Raab-Graham KF, Workman ER, Namjoshi S, et al. (2016) Pushing the threshold: how NMDAR antagonists induce homeostasis through protein synthesis to remedy depression. Brain Research |
| Sosanya NM, Cacheaux LP, Workman ER, et al. (2015) Mammalian Target of Rapamycin (mTOR) Tagging Promotes Dendritic Branch Variability through the Capture of Ca2+/Calmodulin-dependent Protein Kinase II α (CaMKIIα) mRNAs by the RNA-binding Protein HuD. The Journal of Biological Chemistry. 290: 16357-71 |
| Workman ER, Haddick PC, Bush K, et al. (2015) Rapid antidepressants stimulate the decoupling of GABA(B) receptors from GIRK/Kir3 channels through increased protein stability of 14-3-3η. Molecular Psychiatry. 20: 298-310 |
| Most D, Workman E, Harris RA. (2014) Synaptic adaptations by alcohol and drugs of abuse: changes in microRNA expression and mRNA regulation. Frontiers in Molecular Neuroscience. 7: 85 |
| Workman ER, Niere F, Raab-Graham KF. (2013) mTORC1-dependent protein synthesis underlying rapid antidepressant effect requires GABABR signaling. Neuropharmacology. 73: 192-203 |