Margarita Dinamarca
Affiliations: | University of Basel, Basel, Basel-Stadt, Switzerland |
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Dinamarca MC, Colombo L, Brykczynska U, et al. (2024) Transmission-selective muscle pathology induced by the active propagation of mutant huntingtin across the human neuromuscular synapse. Frontiers in Molecular Neuroscience. 16: 1287510 |
Dinamarca MC, Colombo L, Tousiaki NE, et al. (2022) Synaptic and functional alterations in the development of mutant huntingtin expressing hiPSC-derived neurons. Frontiers in Molecular Biosciences. 9: 916019 |
Dinamarca MC, Guzzetti F, Karpova A, et al. (2016) Ring finger protein 10 is a novel synaptonuclear messenger encoding activation of NMDA receptors in hippocampus. Elife. 5 |
Schwenk J, Pérez-Garci E, Schneider A, et al. (2015) Modular composition and dynamics of native GABAB receptors identified by high-resolution proteomics. Nature Neuroscience |
Dinamarca MC, Di Luca M, Godoy JA, et al. (2015) The soluble extracellular fragment of neuroligin-1 targets Aβ oligomers to the postsynaptic region of excitatory synapses. Biochemical and Biophysical Research Communications. 466: 66-71 |
Dinamarca MC, Ríos JA, Inestrosa NC. (2012) Postsynaptic Receptors for Amyloid-β Oligomers as Mediators of Neuronal Damage in Alzheimer's Disease. Frontiers in Physiology. 3: 464 |
Dinamarca MC, Weinstein D, Monasterio O, et al. (2011) The synaptic protein neuroligin-1 interacts with the amyloid β-peptide. Is there a role in Alzheimer's disease? Biochemistry. 50: 8127-37 |
Dinamarca MC, Sagal JP, Quintanilla RA, et al. (2010) Amyloid-beta-Acetylcholinesterase complexes potentiate neurodegenerative changes induced by the Abeta peptide. Implications for the pathogenesis of Alzheimer's disease. Molecular Neurodegeneration. 5: 4 |
Griffith TN, Varela-Nallar L, Dinamarca MC, et al. (2010) Neurobiological effects of Hyperforin and its potential in Alzheimer's disease therapy. Current Medicinal Chemistry. 17: 391-406 |
Dinamarca MC, Arrázola M, Toledo E, et al. (2008) Release of acetylcholinesterase (AChE) from beta-amyloid plaques assemblies improves the spatial memory impairments in APP-transgenic mice. Chemico-Biological Interactions. 175: 142-9 |