J. Lindsay Whitton
Affiliations: | Immunology and Microbiology | Scripps Research Institute, La Jolla, La Jolla, CA, United States |
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Publications
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| Alirezaei M, Flynn CT, Garcia SD, et al. (2020) A food-responsive switch modulates TFEB and autophagy, and determines susceptibility to coxsackievirus infection and pancreatitis. Autophagy. 1-18 |
| Kimura T, Flynn CT, Alirezaei M, et al. (2019) Biphasic and cardiomyocyte-specific IFIT activity protects cardiomyocytes from enteroviral infection. Plos Pathogens. 15: e1007674 |
| Flynn CT, Kimura T, Frimpong-Boateng K, et al. (2017) Immunological and pathological consequences of coxsackievirus RNA persistence in the heart. Virology. 512: 104-112 |
| Hosking MP, Flynn CT, Whitton JL. (2016) Type I IFN Signaling Is Dispensable during Secondary Viral Infection. Plos Pathogens. 12: e1005861 |
| Hosking MP, Flynn CT, Whitton JL. (2016) TCR independent suppression of CD8(+) T cell cytokine production mediated by IFNγ in vivo. Virology. 498: 69-81 |
| Harkins S, Whitton JL. (2016) Chromosomal mapping of the αMHC-MerCreMer transgene in mice reveals a large genomic deletion. Transgenic Research |
| Alirezaei M, Flynn CT, Wood MR, et al. (2015) Coxsackievirus can exploit LC3 in both autophagy-dependent and -independent manners in vivo. Autophagy. 0 |
| Hosking MP, Flynn CT, Whitton JL. (2014) Antigen-specific naive CD8+ T cells produce a single pulse of IFN-γ in vivo within hours of infection, but without antiviral effect. Journal of Immunology (Baltimore, Md. : 1950). 193: 1873-85 |
| Robinson SM, Tsueng G, Sin J, et al. (2014) Coxsackievirus B exits the host cell in shed microvesicles displaying autophagosomal markers. Plos Pathogens. 10: e1004045 |
| Althof N, Harkins S, Kemball CC, et al. (2014) In vivo ablation of type I interferon receptor from cardiomyocytes delays coxsackieviral clearance and accelerates myocardial disease. Journal of Virology. 88: 5087-99 |