Kelly M. Lohr, B.S.
Affiliations: | Emory University, Atlanta, GA | ||
Harvard University, Cambridge, MA, United States |
Area:
VMAT2, dopamine, Parkinson's diseaseGoogle:
"Kelly Lohr"Mean distance: 17.57 (cluster 32) | S | N | B | C | P |
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Publications
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Sarkar S, Bardai F, Olsen AL, et al. (2021) Oligomerization of Lrrk controls actin severing and α-synuclein neurotoxicity in vivo. Molecular Neurodegeneration. 16: 33 |
Sarkar S, Olsen AL, Sygnecka K, et al. (2021) α-synuclein impairs autophagosome maturation through abnormal actin stabilization. Plos Genetics. 17: e1009359 |
Lohr KM, Frost B, Scherzer C, et al. (2020) Biotin rescues mitochondrial dysfunction and neurotoxicity in a tauopathy model. Proceedings of the National Academy of Sciences of the United States of America |
Branco RC, Burkett JP, Black CA, et al. (2020) Vesicular monoamine transporter 2 mediates fear behavior in mice. Genes, Brain, and Behavior. e12634 |
Dunn AR, Stout KA, Ozawa M, et al. (2017) Synaptic vesicle glycoprotein 2C (SV2C) modulates dopamine release and is disrupted in Parkinson disease. Proceedings of the National Academy of Sciences of the United States of America |
Cliburn RA, Dunn AR, Stout KA, et al. (2016) Immunochemical localization of vesicular monoamine transporter 2 (VMAT2) in mouse brain. Journal of Chemical Neuroanatomy |
Lohr KM, Masoud ST, Salahpour A, et al. (2016) Membrane transporters as mediators of synaptic dopamine dynamics: implications for disease. The European Journal of Neuroscience |
Stout KA, Dunn AR, Lohr KM, et al. (2016) Selective enhancement of dopamine release in the ventral pallidum of methamphetamine-sensitized mice. Acs Chemical Neuroscience |
Lohr KM, Chen M, Hoffman CA, et al. (2016) Vesicular monoamine transporter 2 (VMAT2) level regulates MPTP vulnerability and clearance of excess dopamine in mouse striatal terminals. Toxicological Sciences : An Official Journal of the Society of Toxicology |
Trossbach SV, Bader V, Hecher L, et al. (2016) Misassembly of full-length Disrupted-in-Schizophrenia 1 protein is linked to altered dopamine homeostasis and behavioral deficits. Molecular Psychiatry |