Kelly M. Lohr, B.S.

Emory University, Atlanta, GA 
 Harvard University, Cambridge, MA, United States 
VMAT2, dopamine, Parkinson's disease
"Kelly Lohr"
Mean distance: 17.57 (cluster 32)


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Gary W. Miller grad student 2010-2015 Emory
Mel  B. Feany post-doc
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Sarkar S, Bardai F, Olsen AL, et al. (2021) Oligomerization of Lrrk controls actin severing and α-synuclein neurotoxicity in vivo. Molecular Neurodegeneration. 16: 33
Sarkar S, Olsen AL, Sygnecka K, et al. (2021) α-synuclein impairs autophagosome maturation through abnormal actin stabilization. Plos Genetics. 17: e1009359
Lohr KM, Frost B, Scherzer C, et al. (2020) Biotin rescues mitochondrial dysfunction and neurotoxicity in a tauopathy model. Proceedings of the National Academy of Sciences of the United States of America
Branco RC, Burkett JP, Black CA, et al. (2020) Vesicular monoamine transporter 2 mediates fear behavior in mice. Genes, Brain, and Behavior. e12634
Dunn AR, Stout KA, Ozawa M, et al. (2017) Synaptic vesicle glycoprotein 2C (SV2C) modulates dopamine release and is disrupted in Parkinson disease. Proceedings of the National Academy of Sciences of the United States of America
Cliburn RA, Dunn AR, Stout KA, et al. (2016) Immunochemical localization of vesicular monoamine transporter 2 (VMAT2) in mouse brain. Journal of Chemical Neuroanatomy
Lohr KM, Masoud ST, Salahpour A, et al. (2016) Membrane transporters as mediators of synaptic dopamine dynamics: implications for disease. The European Journal of Neuroscience
Stout KA, Dunn AR, Lohr KM, et al. (2016) Selective enhancement of dopamine release in the ventral pallidum of methamphetamine-sensitized mice. Acs Chemical Neuroscience
Lohr KM, Chen M, Hoffman CA, et al. (2016) Vesicular monoamine transporter 2 (VMAT2) level regulates MPTP vulnerability and clearance of excess dopamine in mouse striatal terminals. Toxicological Sciences : An Official Journal of the Society of Toxicology
Trossbach SV, Bader V, Hecher L, et al. (2016) Misassembly of full-length Disrupted-in-Schizophrenia 1 protein is linked to altered dopamine homeostasis and behavioral deficits. Molecular Psychiatry
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