Jean-Luc Balligand
Affiliations: | 1991-1994 | Cardiovascular Medicine | Brigham & Women's Hospital, Harvard Medical School, Boston, MA, United States |
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Parents
Sign in to add mentor| Thomas Michel | post-doc | 1991-1994 | Brigham & Women's Hospital, Harvard Medical School |
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Publications
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| Dufeys C, Daskalopoulos EP, Castanares-Zapatero D, et al. (2021) AMPKα1 deletion in myofibroblasts exacerbates post-myocardial infarction fibrosis by a connexin 43 mechanism. Basic Research in Cardiology. 116: 10 |
| Montiel V, Bella R, Michel LYM, et al. (2020) Inhibition of aquaporin-1 prevents myocardial remodeling by blocking the transmembrane transport of hydrogen peroxide. Science Translational Medicine. 12 |
| Schobesberger S, Wright PT, Poulet C, et al. (2020) β-Adrenoceptor redistribution impairs NO/cGMP/PDE2 signalling in failing cardiomyocytes. Elife. 9 |
| Dubois-Deruy E, Gelinas R, Beauloye C, et al. (2020) Beta 3 adrenoreceptors protect from hypertrophic remodelling through AMP-activated protein kinase and autophagy. Esc Heart Failure |
| Dei Zotti F, Verdoy R, Brusa D, et al. (2020) Redox regulation of nitrosyl-hemoglobin in human erythrocytes. Redox Biology. 34: 101399 |
| Montiel V, Bella R, Michel L, et al. (2020) Inhibition of the water channel aquaporin-1 prevents myocardial remodeling by blocking the transmembrane transport of hydrogen peroxide European Heart Journal. 41 |
| André E, De Pauw A, Verdoy R, et al. (2019) Changes of metabolic phenotype of cardiac progenitor cells during differentiation: Neutral effect of stimulation of AMP-activated protein kinase. Stem Cells and Development |
| Turkieh A, Porouchani S, Beseme O, et al. (2019) Increased clusterin levels after myocardial infarction is due to a defect in protein degradation systems activity. Cell Death & Disease. 10: 608 |
| Maack C, Eschenhagen T, Hamdani N, et al. (2018) Treatments targeting inotropy. European Heart Journal |
| Dei Zotti F, Lobysheva II, Balligand JL. (2018) Nitrosyl-hemoglobin formation in rodent and human venous erythrocytes reflects NO formation from the vasculature in vivo. Plos One. 13: e0200352 |