Julie Quoyer
Affiliations: | Institute for Research in Immunology and Cancer | Université de Montréal, Montréal, Canada |
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| Carr R, Du Y, Quoyer J, et al. (2014) Development and characterization of pepducins as Gs-biased allosteric agonists. The Journal of Biological Chemistry. 289: 35668-84 |
| Armando S, Quoyer J, Lukashova V, et al. (2014) The chemokine CXC4 and CC2 receptors form homo- and heterooligomers that can engage their signaling G-protein effectors and βarrestin. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 28: 4509-23 |
| Quoyer J, Janz JM, Luo J, et al. (2013) Pepducin targeting the C-X-C chemokine receptor type 4 acts as a biased agonist favoring activation of the inhibitory G protein. Proceedings of the National Academy of Sciences of the United States of America. 110: E5088-97 |
| Dalle S, Quoyer J, Varin E, et al. (2011) Roles and regulation of the transcription factor CREB in pancreatic β -cells. Current Molecular Pharmacology. 4: 187-95 |
| Broca C, Quoyer J, Costes S, et al. (2009) beta-Arrestin 1 is required for PAC1 receptor-mediated potentiation of long-lasting ERK1/2 activation by glucose in pancreatic beta-cells. The Journal of Biological Chemistry. 284: 4332-42 |
| Binet V, Duthey B, Lecaillon J, et al. (2007) Common structural requirements for heptahelical domain function in class A and class C G protein-coupled receptors. The Journal of Biological Chemistry. 282: 12154-63 |