Lucas Schirmer
Affiliations: | Ruprecht-Karls-University Heidelberg, Heidelberg, Baden-Württemberg, Germany |
Area:
Neurology, Neurobiology, Neuroimmunology, Neuropathology, Systems BiologyGoogle:
"Lucas Schirmer"Mean distance: (not calculated yet)
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Publications
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| Brenner D, Sieverding K, Srinidhi J, et al. (2024) A TBK1 variant causes autophagolysosomal and motoneuron pathology without neuroinflammation in mice. The Journal of Experimental Medicine. 221 |
| Kapell H, Fazio L, Dyckow J, et al. (2023) Neuron-oligodendrocyte potassium shuttling at nodes of Ranvier protects against inflammatory demyelination. The Journal of Clinical Investigation |
| Trobisch T, Zulji A, Stevens NA, et al. (2022) Cross-regional homeostatic and reactive glial signatures in multiple sclerosis. Acta Neuropathologica |
| Ben Haim L, Schirmer L, Zulji A, et al. (2021) Evidence for glutamine synthetase function in mouse spinal cord oligodendrocytes. Glia |
| Schirmer L, Schafer DP, Bartels T, et al. (2021) Diversity and Function of Glial Cell Types in Multiple Sclerosis. Trends in Immunology |
| Beutel T, Dzimiera J, Kapell H, et al. (2020) Cortical projection neurons as a therapeutic target in multiple sclerosis. Expert Opinion On Therapeutic Targets. 1-14 |
| Schirmer L, Velmeshev D, Holmqvist S, et al. (2019) Neuronal vulnerability and multilineage diversity in multiple sclerosis. Nature |
| Velmeshev D, Schirmer L, Jung D, et al. (2019) Single-cell genomics identifies cell type-specific molecular changes in autism. Science (New York, N.Y.). 364: 685-689 |
| Schirmer L, Möbius W, Zhao C, et al. (2018) Oligodendrocyte-encoded Kir4.1 function is required for axonal integrity. Elife. 7 |
| Kelley KW, Ben Haim L, Schirmer L, et al. (2018) Kir4.1-Dependent Astrocyte-Fast Motor Neuron Interactions Are Required for Peak Strength. Neuron |