Carla Nau

University of Erlangen-Nuremberg, Erlangen, Germany, Erlangen, Bayern, Germany 
Sodium channels; TRP channels; Local anesthetics; Pain plasticity
"Carla Nau"
Mean distance: 21.21 (cluster 22)
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O'Reilly AO, Lattrell A, Miles AJ, et al. (2017) Mutagenesis of the NaChBac sodium channel discloses a functional role for a conserved S6 asparagine. European Biophysics Journal : Ebj
Eberhardt E, Havlicek S, Schmidt D, et al. (2015) Pattern of Functional TTX-Resistant Sodium Channels Reveals a Developmental Stage of Human iPSC- and ESC-Derived Nociceptors. Stem Cell Reports. 5: 305-13
Eberhardt M, Nakajima J, Klinger AB, et al. (2014) Inherited pain: sodium channel Nav1.7 A1632T mutation causes erythromelalgia due to a shift of fast inactivation. The Journal of Biological Chemistry. 289: 1971-80
Bierhaus A, Fleming T, Stoyanov S, et al. (2012) Methylglyoxal modification of Nav1.8 facilitates nociceptive neuron firing and causes hyperalgesia in diabetic neuropathy. Nature Medicine. 18: 926-33
Fischer MJ, Leffler A, Niedermirtl F, et al. (2010) The general anesthetic propofol excites nociceptors by activating TRPV1 and TRPA1 rather than GABAA receptors. The Journal of Biological Chemistry. 285: 34781-92
Choi JS, Cheng X, Foster E, et al. (2010) Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia. Brain : a Journal of Neurology. 133: 1823-35
Wang CF, Gerner P, Schmidt B, et al. (2008) Use of bulleyaconitine A as an adjuvant for prolonged cutaneous analgesia in the rat. Anesthesia and Analgesia. 107: 1397-405
Harrison N, Nau C. (2008) Sensitization of nociceptive ion channels by inhaled anesthetics--a pain in the gas? Molecular Pharmacology. 74: 1180-2
Zimmermann K, Leffler A, Babes A, et al. (2007) Sensory neuron sodium channel Nav1.8 is essential for pain at low temperatures. Nature. 447: 855-8
Leffler A, Reiprich A, Mohapatra DP, et al. (2007) Use-dependent block by lidocaine but not amitriptyline is more pronounced in tetrodotoxin (TTX)-Resistant Nav1.8 than in TTX-sensitive Na+ channels. The Journal of Pharmacology and Experimental Therapeutics. 320: 354-64
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