Fengping Dong
Affiliations: | 2013-2019 | Biology | Penn State University, University Park |
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Sign in to add mentorYingwei Mao | grad student | 2013-2019 | Penn State University, University Park |
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Publications
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McSweeney C, Chen M, Dong F, et al. (2023) Transcriptomic Analyses of Brains of RBM8A Conditional Knockout Mice at Different Developmental Stages Reveal Conserved Signaling Pathways Contributing to Neurodevelopmental Diseases. International Journal of Molecular Sciences. 24 |
Dong F, Mao J, Chen M, et al. (2021) Schizophrenia risk ZNF804A interacts with its associated proteins to modulate dendritic morphology and synaptic development. Molecular Brain. 14: 12 |
McSweeney C, Dong F, Chen M, et al. (2020) Full function of exon junction complex factor, Rbm8a, is critical for interneuron development. Translational Psychiatry. 10: 379 |
Zhou Y, Dong F, Mao Y. (2018) Control of CNS functions by RNA-binding proteins in neurological diseases. Current Pharmacology Reports. 4: 301-313 |
Cheng G, Li W, Ha L, et al. (2018) Self-Assembly of Extracellular Vesicle-like Metal-Organic Framework Nanoparticles for Protection and Intracellular Delivery of Biofunctional Proteins. Journal of the American Chemical Society |
Lai J, Li S, Shi X, et al. (2017) Displacement and hybridization reactions in aptamer-functionalized hydrogels for biomimetic protein release and signal transduction. Chemical Science. 8: 7306-7311 |
Deng D, Jian C, Lei L, et al. (2017) A prenatal interruption of DISC1 function in the brain exhibits a lasting impact on adult behaviors, brain metabolism, and interneuron development. Oncotarget. 8: 84798-84817 |
Dong F, Xie K, Chen Y, et al. (2016) Polycistronic tRNA and CRISPR guide-RNA enables highly efficient multiplexed genome engineering in human cells. Biochemical and Biophysical Research Communications |
Zou D, Zhou Y, Liu L, et al. (2016) Transient enhancement of proliferation of neural progenitors and impairment of their long-term survival in p25 transgenic mice. Oncotarget |
Dong F, Jiang J, McSweeney C, et al. (2016) Deletion of CTNNB1 in inhibitory circuitry contributes to autism-associated behavioral defects. Human Molecular Genetics |