Veronica Vizuet Galvan
Affiliations: | 2001 | University of California, Irvine, Irvine, CA |
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"Veronica Galvan"Mean distance: 15.19 (cluster 19) | S | N | B | C | P |
Parents
Sign in to add mentorNorman M. Weinberger | grad student | 2001 | UC Irvine | |
(Long-term stability of frequency tuning and consolidation of learning -induced receptive field plasticity in the primary auditory cortex.) |
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Publications
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Chocron ES, Munkácsy E, Kim HS, et al. (2022) Genetic and pharmacologic proteasome augmentation ameliorates Alzheimer's-like pathology in mouse and fly APP overexpression models. Science Advances. 8: eabk2252 |
Dorigatti AO, Riordan R, Yu Z, et al. (2022) Brain cellular senescence in mouse models of Alzheimer's disease. Geroscience |
Qiu S, Palavicini JP, Wang J, et al. (2021) Adult-onset CNS myelin sulfatide deficiency is sufficient to cause Alzheimer's disease-like neuroinflammation and cognitive impairment. Molecular Neurodegeneration. 16: 64 |
Van Skike CE, Hussong SA, Hernandez SF, et al. (2021) mTOR attenuation with rapamycin reverses neurovascular uncoupling and memory deficits in mice modeling Alzheimer's disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Van Skike CE, Lin AL, Roberts Burbank R, et al. (2019) mTOR drives cerebrovascular, synaptic, and cognitive dysfunction in normative aging. Aging Cell. e13057 |
Kaeberlein M, Galvan V. (2019) Rapamycin and Alzheimer's disease: Time for a clinical trial? Science Translational Medicine. 11 |
Hernandez SF, Skike CEV, DeRosa N, et al. (2019) Mtor Promotes Bbb Breakdown In A Model Of Alzheimer’S Disease Innovation in Aging. 3 |
Banh A, Pomilio C, Hussong SA, et al. (2019) Mtor-Driven Alterations In The Brain Microvascular Proteome In Alzheimer’S Disease Innovation in Aging. 3 |
Olson A, Hussong SA, Kayed R, et al. (2019) TAU-INDUCED ASTROCYTE SENESCENCE: A NOVEL MECHANISM FOR NEURONAL DYSFUNCTION IN ALZHEIMER’S DISEASE Innovation in Aging. 3: S91-S92 |
Hussong SA, Skike CEV, Olson AB, et al. (2019) P4-148: Propagation Of Soluble Tau Aggregates In Brain Microvascular Endothelial Cells Promotes Cellular Senescence/Sasp And Blocks Enos Activation Alzheimers & Dementia. 15 |