Misha Zilberter
Affiliations: | Neurological Disease | Gladstone Institutes |
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| Rao A, Chen N, Kim MJ, et al. (2024) Microglia depletion reduces human neuronal APOE4-related pathologies in a chimeric Alzheimer's disease model. Cell Stem Cell |
| Koutsodendris N, Blumenfeld J, Agrawal A, et al. (2023) APOE4-promoted gliosis and degeneration in tauopathy are ameliorated by pharmacological inhibition of HMGB1 release. Cell Reports. 113252 |
| Zilberter Y, Tabuena DR, Zilberter M. (2023) NOX-induced oxidative stress is a primary trigger of major neurodegenerative disorders. Progress in Neurobiology. 231: 102539 |
| Jang SS, Tabuena DR, Grone B, et al. (2023) Neuronal apoE4 induces early hyperexcitability in select populations of hippocampal neurons by altering Nell2 expression. Biorxiv : the Preprint Server For Biology |
| Koutsodendris N, Blumenfeld J, Agrawal A, et al. (2023) Neuronal APOE4 removal protects against tau-mediated gliosis, neurodegeneration and myelin deficits. Nature Aging. 3: 275-296 |
| Li H, Guglielmetti C, Sei YJ, et al. (2023) Neurons require glucose uptake and glycolysis in vivo. Cell Reports. 42: 112335 |
| Zilberter Y, Popova I, Zilberter M. (2022) Unifying mechanism behind the onset of acquired epilepsy. Trends in Pharmacological Sciences. 43: 87-96 |
| Taubes A, Nova P, Zalocusky KA, et al. (2021) Experimental and real-world evidence supporting the computational repurposing of bumetanide for -related Alzheimer's disease. Nature Aging. 1: 932-947 |
| Malkov A, Popova I, Ivanov A, et al. (2021) Aβ initiates brain hypometabolism, network dysfunction and behavioral abnormalities via NOX2-induced oxidative stress in mice. Communications Biology. 4: 1054 |
| Najm R, Zalocusky KA, Zilberter M, et al. (2020) In Vivo Chimeric Alzheimer's Disease Modeling of Apolipoprotein E4 Toxicity in Human Neurons. Cell Reports. 32: 107962 |