David W. Allison
Affiliations: | 2010 | Physiology and Developmental Biology | Brigham Young University, Provo, UT, United States |
Area:
addictionGoogle:
"David Allison"Mean distance: 15.64 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentor| Scott Steffensen | grad student | 2010 | BYU | |
| (Cocaine and mefloquine-induced acute effects in ventral tegmental area dopamine and GABA neurons.) | ||||
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Publications
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| Steffensen SC, Bradley KD, Hansen DM, et al. (2011) The role of connexin-36 gap junctions in alcohol intoxication and consumption. Synapse (New York, N.Y.). 65: 695-707 |
| Allison DW, Wilcox RS, Ellefsen KL, et al. (2011) Mefloquine effects on ventral tegmental area dopamine and GABA neuron inhibition: a physiologic role for connexin-36 GAP junctions. Synapse (New York, N.Y.). 65: 804-13 |
| Vargas-Perez H, Ting-A Kee R, Walton CH, et al. (2009) Ventral tegmental area BDNF induces an opiate-dependent-like reward state in naive rats. Science (New York, N.Y.). 324: 1732-4 |
| Ludlow KH, Bradley KD, Allison DW, et al. (2009) Acute and chronic ethanol modulate dopamine D2-subtype receptor responses in ventral tegmental area GABA neurons. Alcoholism, Clinical and Experimental Research. 33: 804-11 |
| Steffensen SC, Taylor SR, Horton ML, et al. (2008) Cocaine disinhibits dopamine neurons in the ventral tegmental area via use-dependent blockade of GABA neuron voltage-sensitive sodium channels. The European Journal of Neuroscience. 28: 2028-40 |
| Steffensen SC, Jones MD, Hales K, et al. (2006) Dehydroepiandrosterone sulfate and estrone sulfate reduce GABA-recurrent inhibition in the hippocampus via muscarinic acetylcholine receptors. Hippocampus. 16: 1080-90 |
| Allison DW, Ohran AJ, Stobbs SH, et al. (2006) Connexin-36 gap junctions mediate electrical coupling between ventral tegmental area GABA neurons. Synapse (New York, N.Y.). 60: 20-31 |
| Stobbs SH, Ohran AJ, Lassen MB, et al. (2004) Ethanol suppression of ventral tegmental area GABA neuron electrical transmission involves N-methyl-D-aspartate receptors. The Journal of Pharmacology and Experimental Therapeutics. 311: 282-9 |