Marta Zamarbide
Affiliations: | 2014- | Pharmacology | The George Washington University, Washington, DC, United States |
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| Abellanas MA, Zamarbide M, Basurco L, et al. (2019) Midbrain microglia mediate a specific immunosuppressive response under inflammatory conditions. Journal of Neuroinflammation. 16: 233 |
| Zamarbide M, Mossa A, Muñoz-Llancao P, et al. (2018) Male-Specific cAMP Signaling in the Hippocampus Controls Spatial Memory Deficits in a Mouse Model of Autism and Intellectual Disability. Biological Psychiatry |
| Zamarbide M, Gil-Bea FJ, Bannenberg P, et al. (2018) Maternal imprinting on cognition markers of wild type and transgenic Alzheimer's disease model mice. Scientific Reports. 8: 6434 |
| Zamarbide M, Oaks AW, Pond HL, et al. (2018) Loss of the Intellectual Disability and Autism Geneand Its HomologDifferentially Affect Spatial Memory, Anxiety, and Hyperactivity. Frontiers in Genetics. 9: 65 |
| Franco R, Martínez-Pinilla E, Navarro G, et al. (2017) Potential of GPCRs to modulate MAPK and mTOR pathways in Alzheimer's disease. Progress in Neurobiology |
| Choi SB, Zamarbide M, Manzini MC, et al. (2016) Tapered-Tip Capillary Electrophoresis Nano-Electrospray Ionization Mass Spectrometry for Ultrasensitive Proteomics: the Mouse Cortex. Journal of the American Society For Mass Spectrometry |
| Oaks AW, Zamarbide M, Tambunan DE, et al. (2016) Cc2d1a Loss of Function Disrupts Functional and Morphological Development in Forebrain Neurons Leading to Cognitive and Social Deficits. Cerebral Cortex (New York, N.Y. : 1991) |
| López-González I, Pérez-Mediavilla A, Zamarbide M, et al. (2016) Limited Unfolded Protein Response and Inflammation in Neuroserpinopathy. Journal of Neuropathology and Experimental Neurology |
| Zamarbide M, Etayo-Labiano I, Ricobaraza A, et al. (2014) GPR40 activation leads to CREB and ERK phosphorylation in primary cultures of neurons from the mouse CNS and in human neuroblastoma cells. Hippocampus. 24: 733-9 |
| Cuadrado-Tejedor M, Cabodevilla JF, Zamarbide M, et al. (2013) Age-related mitochondrial alterations without neuronal loss in the hippocampus of a transgenic model of Alzheimer's disease. Current Alzheimer Research. 10: 390-405 |