Sarah Ann Busch

Affiliations: 
2009 Case Western Reserve University, Cleveland Heights, OH, United States 
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Jerry Silver grad student 2009 Case Western
 (Cellular and molecular strategies to overcome macrophage-mediated axonal dieback after spinal cord injury.)
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Publications

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Jones JM, DePaul M, Gregory CR, et al. (2018) Multipotent adult progenitor cells, but not TIMP3, increase tissue sparing and reduce urological complications following spinal cord injury. Journal of Neurotrauma
Jones J, DePaul M, Zhu M, et al. (2017) MP41-06 MULTIPOTENT ADULT PROGENITOR CELL TREATMENT PROMOTES BLADDER FUNCTION RECOVERY AND DECREASES MORBIDITY AND DURATION OF URINARY TRACT INFECTION IN SPINAL CORD INJURED RATS Journal of Urology. 197
DePaul MA, Palmer M, Lang BT, et al. (2015) Intravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury. Scientific Reports. 5: 16795
Lang BT, Cregg JM, DePaul MA, et al. (2015) Modulation of the proteoglycan receptor PTPσ promotes recovery after spinal cord injury. Nature. 518: 404-8
Filous AR, Tran A, Howell CJ, et al. (2014) Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 16369-84
Reading JL, Yang JH, Sabbah S, et al. (2013) Clinical-grade multipotent adult progenitor cells durably control pathogenic T cell responses in human models of transplantation and autoimmunity. Journal of Immunology (Baltimore, Md. : 1950). 190: 4542-52
Busch SA, Hamilton JA, Horn KP, et al. (2011) Multipotent adult progenitor cells prevent macrophage-mediated axonal dieback and promote regrowth after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 31: 944-53
Busch SA, Horn KP, Cuascut FX, et al. (2010) Adult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 255-65
Silver J, Horn KP, Busch SA, et al. (2010) Axonal regeneration: Role of the extracellular matrix and the glial scar Encyclopedia of Neuroscience. 1173-1180
Shen Y, Tenney AP, Busch SA, et al. (2009) PTPsigma is a receptor for chondroitin sulfate proteoglycan, an inhibitor of neural regeneration. Science (New York, N.Y.). 326: 592-6
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