Jennifer A. Bendiske, Ph.D. - Publications
Affiliations: | University of Connecticut, Storrs, CT, United States |
Area:
Neuroprotection in models of Alzheimer-type pathogenesisYear | Citation | Score | |||
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2007 | Kanju PM, Parameshwaran K, Vaithianathan T, Sims CM, Huggins K, Bendiske J, Ryzhikov S, Bahr BA, Suppiramaniam V. Lysosomal dysfunction produces distinct alterations in synaptic alpha-amino-3-hydroxy-5-methylisoxazolepropionic acid and N-methyl-D-aspartate receptor currents in hippocampus. Journal of Neuropathology and Experimental Neurology. 66: 779-88. PMID 17805008 DOI: 10.1097/nen.0b013e3181461ae7 | 0.648 | |||
2007 | Butler D, Bendiske J, Michaelis ML, Karanian DA, Bahr BA. Microtubule-stabilizing agent prevents protein accumulation-induced loss of synaptic markers. European Journal of Pharmacology. 562: 20-7. PMID 17336290 DOI: 10.1016/J.Ejphar.2007.01.053 | 0.686 | |||
2003 | Bendiske J, Bahr BA. Lysosomal activation is a compensatory response against protein accumulation and associated synaptopathogenesis--an approach for slowing Alzheimer disease? Journal of Neuropathology and Experimental Neurology. 62: 451-63. PMID 12769185 DOI: 10.1093/Jnen/62.5.451 | 0.711 | |||
2002 | Bahr BA, Bendiske J. The neuropathogenic contributions of lysosomal dysfunction. Journal of Neurochemistry. 83: 481-9. PMID 12390510 DOI: 10.1046/J.1471-4159.2002.01192.X | 0.617 | |||
2002 | Bendiske J, Caba E, Brown QB, Bahr BA. Intracellular deposition, microtubule destabilization, and transport failure: an "early" pathogenic cascade leading to synaptic decline. Journal of Neuropathology and Experimental Neurology. 61: 640-50. PMID 12125743 DOI: 10.1093/Jnen/61.7.640 | 0.626 | |||
2002 | Bahr BA, Bendiske J, Brown QB, Munirathinam S, Caba E, Rudin M, Urwyler S, Sauter A, Rogers G. Survival signaling and selective neuroprotection through glutamatergic transmission. Experimental Neurology. 174: 37-47. PMID 11869032 DOI: 10.1006/Exnr.2001.7852 | 0.602 | |||
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