| Year |
Citation |
Score |
| 2022 |
Jiang K, Mondal AK, Adlakha YK, Gumerson J, Aponte A, Gieser L, Kim JW, Boleda A, Brooks MJ, Nellissery J, Fox DA, Balaban R, Covian R, Swaroop A. Multi-omics analyses reveal early metabolic imbalance and mitochondrial stress in neonatal photoreceptors leading to cell death in Pde6brd1/rd1 mouse model of retinal degeneration. Human Molecular Genetics. PMID 35075486 DOI: 10.1093/hmg/ddac013 |
0.395 |
|
| 2016 |
Shen XF, Huang P, Fox DA, Lin Y, Zhao ZH, Wang W, Wang JY, Liu XQ, Chen JY, Luo WJ. Adult lead exposure increases blood-retinal permeability: A risk factor for retinal vascular disease. Neurotoxicology. PMID 27663850 DOI: 10.1016/j.neuro.2016.09.013 |
0.405 |
|
| 2016 |
Poché RA, Zhang M, Rueda EM, Tong X, McElwee ML, Wong L, Hsu CW, Dejosez M, Burns AR, Fox DA, Martin JF, Zwaka TP, Dickinson ME. RONIN Is an Essential Transcriptional Regulator of Genes Required for Mitochondrial Function in the Developing Retina. Cell Reports. PMID 26876175 DOI: 10.1016/J.Celrep.2016.01.039 |
0.387 |
|
| 2015 |
Fox DA, Rueda EM. Photoreceptor Mitochondrial Reserve Capacity: Is It the Etiology for Differential Compartmental Vulnerability? Investigative Ophthalmology & Visual Science. 56: 8437. PMID 26747774 DOI: 10.1167/iovs.15-18662 |
0.36 |
|
| 2015 |
Fox DA. Retinal and visual system: occupational and environmental toxicology. Handbook of Clinical Neurology. 131: 325-40. PMID 26563796 DOI: 10.1016/B978-0-444-62627-1.00017-2 |
0.389 |
|
| 2015 |
Fox DA. Gestational lead exposure in humans and experimental animals: Novel functional and morphological phenotype and late-onset retinal degeneration Neurotoxicology and Teratology. 49: 113-114. DOI: 10.1016/J.NTT.2015.04.049 |
0.388 |
|
| 2013 |
Ahmedli NB, Gribanova Y, Njoku CC, Naidu A, Young A, Mendoza E, Yamashita CK, Ozgül RK, Johnson JE, Fox DA, Farber DB. Dynamics of the rhomboid-like protein RHBDD2 expression in mouse retina and involvement of its human ortholog in retinitis pigmentosa. The Journal of Biological Chemistry. 288: 9742-54. PMID 23386608 DOI: 10.1074/Jbc.M112.419960 |
0.393 |
|
| 2012 |
Perkins GA, Scott R, Perez A, Ellisman MH, Johnson JE, Fox DA. Bcl-xL-mediated remodeling of rod and cone synaptic mitochondria after postnatal lead exposure: electron microscopy, tomography and oxygen consumption. Molecular Vision. 18: 3029-48. PMID 23288995 |
0.369 |
|
| 2012 |
De Groot D, De Groot D, Fox D, Waalkens I, Lammers J. Fetal exposure to MAM in rats induces life-lasting non-progressive neural impairment Toxicology Letters. 211: S93-S94. DOI: 10.1016/J.TOXLET.2012.03.352 |
0.305 |
|
| 2011 |
Fox DA, Hamilton WR, Johnson JE, Xiao W, Chaney S, Mukherjee S, Miller DB, O'Callaghan JP. Gestational lead exposure selectively decreases retinal dopamine amacrine cells and dopamine content in adult mice. Toxicology and Applied Pharmacology. 256: 258-67. PMID 21703292 DOI: 10.1016/j.taap.2011.05.021 |
0.381 |
|
| 2011 |
Giddabasappa A, Hamilton WR, Chaney S, Xiao W, Johnson JE, Mukherjee S, Fox DA. Low-level gestational lead exposure increases retinal progenitor cell proliferation and rod photoreceptor and bipolar cell neurogenesis in mice. Environmental Health Perspectives. 119: 71-7. PMID 20840909 DOI: 10.1289/ehp.1002524 |
0.391 |
|
| 2010 |
Fox DA, Opanashuk L, Zharkovsky A, Weiss B. Gene-chemical interactions in the developing mammalian nervous system: Effects on proliferation, neurogenesis and differentiation. Neurotoxicology. 31: 589-97. PMID 20381523 DOI: 10.1016/j.neuro.2010.03.007 |
0.359 |
|
| 2008 |
Fox DA, Kala SV, Hamilton WR, Johnson JE, O'Callaghan JP. Low-level human equivalent gestational lead exposure produces supernormal scotopic electroretinograms, increased retinal neurogenesis, and decreased retinal dopamine utilization in rats. Environmental Health Perspectives. 116: 618-25. PMID 18470321 DOI: 10.1289/ehp.11268 |
0.355 |
|
| 2007 |
Johnson JE, Perkins GA, Giddabasappa A, Chaney S, Xiao W, White AD, Brown JM, Waggoner J, Ellisman MH, Fox DA. Spatiotemporal regulation of ATP and Ca2+ dynamics in vertebrate rod and cone ribbon synapses. Molecular Vision. 13: 887-919. PMID 17653034 |
0.375 |
|
| 2007 |
Fox DA, Perkins GA, Johnson JE, Dixit A, Ellisman MH. 15 Differential susceptibility of rod photoreceptor synaptic and non-synaptic mitochondria to divalent cation exposure: Neuroprotection by Bcl-xL overexpression Mitochondrion. 7: 408. DOI: 10.1016/J.MITO.2007.08.019 |
0.34 |
|
| 2004 |
Perkins GA, Ellisman MH, Fox DA. The structure-function correlates of mammalian rod and cone photoreceptor mitochondria: observations and unanswered questions. Mitochondrion. 4: 695-703. PMID 16120425 DOI: 10.1016/j.mito.2004.07.020 |
0.464 |
|
| 2003 |
Fox DA, Poblenz AT, He L, Harris JB, Medrano CJ. Pharmacological strategies to block rod photoreceptor apoptosis caused by calcium overload: A mechanistic target-site approach to neuroprotection. European Journal of Ophthalmology. 13: 44-56. PMID 28252798 DOI: 10.5301/EJO.2008.5701 |
0.79 |
|
| 2003 |
Fox DA, Poblenz AT, He L, Harris JB, Medrano CJ. Pharmacological strategies to block rod photoreceptor apoptosis caused by calcium overload: a mechanistic target-site approach to neuroprotection. European Journal of Ophthalmology. 13: S44-56. PMID 12749677 |
0.778 |
|
| 2003 |
He L, Perkins GA, Poblenz AT, Harris JB, Hung M, Ellisman MH, Fox DA. Bcl-xL overexpression blocks bax-mediated mitochondrial contact site formation and apoptosis in rod photoreceptors of lead-exposed mice. Proceedings of the National Academy of Sciences of the United States of America. 100: 1022-7. PMID 12540825 DOI: 10.1073/Pnas.0333594100 |
0.786 |
|
| 2000 |
He L, Poblenz AT, Medrano CJ, Fox DA. Lead and calcium produce rod photoreceptor cell apoptosis by opening the mitochondrial permeability transition pore. The Journal of Biological Chemistry. 275: 12175-84. PMID 10766853 DOI: 10.1074/Jbc.275.16.12175 |
0.791 |
|
| 2000 |
Söderpalm AK, Fox DA, Karlsson JO, van Veen T. Retinoic acid produces rod photoreceptor selective apoptosis in developing mammalian retina. Investigative Ophthalmology & Visual Science. 41: 937-47. PMID 10711716 |
0.346 |
|
| 1999 |
Fox DA, Poblenz AT, He L. Calcium overload triggers rod photoreceptor apoptotic cell death in chemical-induced and inherited retinal degenerations. Annals of the New York Academy of Sciences. 893: 282-5. PMID 10672249 DOI: 10.1111/J.1749-6632.1999.Tb07837.X |
0.776 |
|
| 1999 |
Timmers AM, Fox DA, He L, Hansen RM, Fulton AB. Rod photoreceptor maturation does not vary with retinal eccentricity in mammalian retina. Current Eye Research. 18: 393-402. PMID 10435825 DOI: 10.1076/ceyr.18.6.393.5263 |
0.413 |
|
| 1998 |
Fox DA, Srivastava D, Poblenz A, He L. Lead-induced Alterations in Gene Expression and Activity of Retinal cGMP PDE Results in Calcium Overload and Rod-selective Apoptosis. Toxicology in Vitro : An International Journal Published in Association With Bibra. 12: 597-8. PMID 20654449 DOI: 10.1016/S0887-2333(98)00044-7 |
0.78 |
|
| 1998 |
Fox DA, He L, Poblenz AT, Medrano CJ, Blocker YS, Srivastava D. Lead-induced alterations in retinal cGMP phosphodiesterase trigger calcium overload, mitochondrial dysfunction and rod photoreceptor apoptosis. Toxicology Letters. 102: 359-61. PMID 10022279 DOI: 10.1016/S0378-4274(98)00232-X |
0.799 |
|
| 1998 |
Adamus G, Machnicki M, Elerding H, Sugden B, Blocker YS, Fox DA. Antibodies to recoverin induce apoptosis of photoreceptor and bipolar cells in vivo. Journal of Autoimmunity. 11: 523-33. PMID 9802939 DOI: 10.1006/jaut.1998.0221 |
0.384 |
|
| 1998 |
Fox D, Srivastava D, Poblenz A, He L. Lead-induced alterations in retinal cGMP PDE gene expression and activity result in cell-specific apoptosis during development Toxicology Letters. 95: 18. DOI: 10.1016/S0378-4274(98)80071-4 |
0.775 |
|
| 1995 |
Fox DA, Srivastava D. Molecular mechanism of the lead-induced inhibition of rod cGMP phosphodiesterase. Toxicology Letters. 82: 263-70. PMID 8597063 DOI: 10.1016/0378-4274(95)03560-5 |
0.326 |
|
| 1995 |
Srivastava D, Hurwitz RL, Fox DA. Lead- and calcium-mediated inhibition of bovine rod cGMP phosphodiesterase: interactions with magnesium. Toxicology and Applied Pharmacology. 134: 43-52. PMID 7676457 DOI: 10.1006/taap.1995.1167 |
0.403 |
|
| 1995 |
Medrano CJ, Fox DA. Oxygen consumption in the rat outer and inner retina: light- and pharmacologically-induced inhibition. Experimental Eye Research. 61: 273-84. PMID 7556491 DOI: 10.1016/S0014-4835(05)80122-8 |
0.411 |
|
| 1994 |
Medrano CJ, Fox DA. Substrate-dependent effects of calcium on rat retinal mitochondrial respiration: physiological and toxicological studies. Toxicology and Applied Pharmacology. 125: 309-21. PMID 8171438 DOI: 10.1006/taap.1994.1077 |
0.421 |
|
| 1992 |
Fox DA, Katz LM. Developmental lead exposure selectively alters the scotopic ERG component of dark and light adaptation and increases rod calcium content. Vision Research. 32: 249-55. PMID 1574840 DOI: 10.1016/0042-6989(92)90134-5 |
0.386 |
|
| 1991 |
Smith EL, Fox DA, Duncan GC. Refractive-error changes in kitten eyes produced by chronic on-channel blockade. Vision Research. 31: 833-44. PMID 2035267 DOI: 10.1016/0042-6989(91)90150-4 |
0.325 |
|
| 1991 |
Fox DA, Katz LM, Farber DB. Low level developmental lead exposure decreases the sensitivity, amplitude and temporal resolution of rods. Neurotoxicology. 12: 641-54. PMID 1665551 |
0.312 |
|
| 1991 |
Fox DA, Rubinstein SD, Hsu P. Developmental lead exposure inhibits adult rat retinal, but not kidney, Na+,K(+)-ATPase. Toxicology and Applied Pharmacology. 109: 482-93. PMID 1649499 DOI: 10.1016/0041-008X(91)90011-3 |
0.374 |
|
| 1989 |
Fox DA, Rubinstein SD. Age-related changes in retinal sensitivity, rhodopsin content and rod outer segment length in hooded rats following low-level lead exposure during development. Experimental Eye Research. 48: 237-49. PMID 2924811 DOI: 10.1016/S0014-4835(89)80073-9 |
0.386 |
|
| 1988 |
Fox DA, Farber DB. Rods are selectively altered by lead: I. Electrophysiology and biochemistry. Experimental Eye Research. 46: 597-611. PMID 2898378 DOI: 10.1016/S0014-4835(88)80016-2 |
0.405 |
|
| 1988 |
Fox DA, Chu LW. Rods are selectively altered by lead: II. Ultrastructure and quantitative histology. Experimental Eye Research. 46: 613-25. PMID 2838312 DOI: 10.1016/S0014-4835(88)80017-4 |
0.495 |
|
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