Year |
Citation |
Score |
2015 |
Zhang Z, David G. Stimulation-induced Ca(2+) influx at nodes of Ranvier in mouse peripheral motor axons. The Journal of Physiology. PMID 26365250 DOI: 10.1113/JP271207 |
0.565 |
|
2014 |
Barrett EF, Barrett JN, David G. Dysfunctional mitochondrial Ca(2+) handling in mutant SOD1 mouse models of fALS: integration of findings from motor neuron somata and motor terminals. Frontiers in Cellular Neuroscience. 8: 184. PMID 25071445 DOI: 10.3389/fncel.2014.00184 |
0.764 |
|
2012 |
Nguyen KT, Zhang Z, Barrett EF, David G. Morphological and functional changes in innervation of a fast forelimb muscle in SOD1-G85R mice. Neurobiology of Disease. 48: 399-408. PMID 22813866 DOI: 10.1016/J.Nbd.2012.07.011 |
0.769 |
|
2012 |
Talbot JD, David G, Barrett EF, Barrett JN. Calcium dependence of damage to mouse motor nerve terminals following oxygen/glucose deprivation. Experimental Neurology. 234: 95-104. PMID 22206924 DOI: 10.1016/J.Expneurol.2011.12.020 |
0.807 |
|
2011 |
Barrett EF, Barrett JN, David G. Mitochondria in motor nerve terminals: function in health and in mutant superoxide dismutase 1 mouse models of familial ALS. Journal of Bioenergetics and Biomembranes. 43: 581-6. PMID 22089637 DOI: 10.1007/s10863-011-9392-1 |
0.785 |
|
2011 |
Nguyen KT, Barrett JN, GarcÃa-Chacón L, David G, Barrett EF. Repetitive nerve stimulation transiently opens the mitochondrial permeability transition pore in motor nerve terminals of symptomatic mutant SOD1 mice. Neurobiology of Disease. 42: 381-90. PMID 21310237 DOI: 10.1016/J.Nbd.2011.01.031 |
0.81 |
|
2010 |
Zhang Z, Nguyen KT, Barrett EF, David G. Vesicular ATPase inserted into the plasma membrane of motor terminals by exocytosis alkalinizes cytosolic pH and facilitates endocytosis. Neuron. 68: 1097-108. PMID 21172612 DOI: 10.1016/J.Neuron.2010.11.035 |
0.764 |
|
2009 |
Nguyen KT, GarcÃa-Chacón LE, Barrett JN, Barrett EF, David G. The Psi(m) depolarization that accompanies mitochondrial Ca2+ uptake is greater in mutant SOD1 than in wild-type mouse motor terminals. Proceedings of the National Academy of Sciences of the United States of America. 106: 2007-11. PMID 19174508 DOI: 10.1073/Pnas.0810934106 |
0.82 |
|
2008 |
Talbot JD, Barrett JN, Barrett EF, David G. Rapid, stimulation-induced reduction of C12-resorufin in motor nerve terminals: linkage to mitochondrial metabolism. Journal of Neurochemistry. 105: 807-19. PMID 18205748 DOI: 10.1111/J.1471-4159.2007.05176.X |
0.797 |
|
2007 |
David G, Nguyen K, Barrett EF. Early vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice. Experimental Neurology. 204: 411-20. PMID 17292357 DOI: 10.1016/J.Expneurol.2006.12.021 |
0.734 |
|
2007 |
Talbot J, Barrett JN, Barrett EF, David G. Stimulation-induced changes in NADH fluorescence and mitochondrial membrane potential in lizard motor nerve terminals. The Journal of Physiology. 579: 783-98. PMID 17218351 DOI: 10.1113/jphysiol.2006.126383 |
0.824 |
|
2006 |
GarcÃa-Chacón LE, Nguyen KT, David G, Barrett EF. Extrusion of Ca2+ from mouse motor terminal mitochondria via a Na+-Ca2+ exchanger increases post-tetanic evoked release. The Journal of Physiology. 574: 663-75. PMID 16613870 DOI: 10.1113/Jphysiol.2006.110841 |
0.827 |
|
2003 |
Talbot JD, David G, Barrett EF. Inhibition of mitochondrial Ca2+ uptake affects phasic release from motor terminals differently depending on external [Ca2+]. Journal of Neurophysiology. 90: 491-502. PMID 12672777 DOI: 10.1152/Jn.00012.2003 |
0.814 |
|
2003 |
David G, Talbot J, Barrett EF. Quantitative estimate of mitochondrial [Ca2+] in stimulated motor nerve terminals. Cell Calcium. 33: 197-206. PMID 12600806 DOI: 10.1016/S0143-4160(02)00229-4 |
0.809 |
|
2003 |
David G, Barrett EF. Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals. The Journal of Physiology. 548: 425-38. PMID 12588898 DOI: 10.1113/jphysiol.2002.035196 |
0.79 |
|
2000 |
David G, Barrett EF. Stimulation-evoked increases in cytosolic [Ca(2+)] in mouse motor nerve terminals are limited by mitochondrial uptake and are temperature-dependent. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 20: 7290-6. PMID 11007886 DOI: 10.1523/Jneurosci.20-19-07290.2000 |
0.8 |
|
1999 |
Howard MJ, David G, Barrett JN. Resealing of transected myelinated mammalian axons in vivo: evidence for involvement of calpain. Neuroscience. 93: 807-15. PMID 10465464 DOI: 10.1016/S0306-4522(99)00195-5 |
0.414 |
|
1999 |
David G. Mitochondrial clearance of cytosolic Ca2+ in stimulated lizard motor nerve terminals proceeds without progressive elevation of mitochondrial matrix [Ca2+] Journal of Neuroscience. 19: 7495-7506. PMID 10460256 DOI: 10.1523/Jneurosci.19-17-07495.1999 |
0.589 |
|
1998 |
David G, Barrett JN, Barrett EF. Evidence that mitochondria buffer physiological Ca2+ loads in lizard motor nerve terminals. The Journal of Physiology. 509: 59-65. PMID 9547381 DOI: 10.1111/j.1469-7793.1998.059bo.x |
0.789 |
|
1997 |
David G, Barrett JN, Barrett EF. Stimulation-induced changes in [Ca2+] in lizard motor nerve terminals. The Journal of Physiology. 504: 83-96. PMID 9350620 DOI: 10.1111/j.1469-7793.1997.083bf.x |
0.769 |
|
1997 |
David G, Barrett JN, Barrett EF. Spatiotemporal gradients of intra-axonal [Na+] after transection and resealing in lizard peripheral myelinated axons. The Journal of Physiology. 498: 295-307. PMID 9032679 DOI: 10.1113/Jphysiol.1997.Sp021858 |
0.683 |
|
1995 |
David G, Modney B, Scappaticci KA, Barrett JN, Barrett EF. Electrical and morphological factors influencing the depolarizing after-potential in rat and lizard myelinated axons. The Journal of Physiology. 489: 141-57. PMID 8583398 DOI: 10.1113/Jphysiol.1995.Sp021037 |
0.684 |
|
1993 |
Morita K, David G, Barrett JN, Barrett EF. Posttetanic hyperpolarization produced by electrogenic Na(+)-K+ pump in lizard axons impaled near their motor terminals. Journal of Neurophysiology. 70: 1874-84. PMID 8294960 DOI: 10.1152/Jn.1993.70.5.1874 |
0.726 |
|
1993 |
David G, Barrett JN, Barrett EF. Activation of internodal potassium conductance in rat myelinated axons. The Journal of Physiology. 472: 177-202. PMID 8145140 DOI: 10.1113/Jphysiol.1993.Sp019942 |
0.706 |
|
1992 |
David G, Barrett JN, Barrett EF. Evidence that action potentials activate an internodal potassium conductance in lizard myelinated axons. The Journal of Physiology. 445: 277-301. PMID 1501136 DOI: 10.1113/Jphysiol.1992.Sp018924 |
0.705 |
|
1986 |
Adler EM, Yaari Y, David G, Selzer ME. Frequency-dependent action of phenytoin on lamprey spinal axons. Brain Research. 362: 271-80. PMID 3942876 DOI: 10.1016/0006-8993(86)90451-8 |
0.604 |
|
1986 |
David G, Selzer ME, Yaari Y. Activity-dependent depression of nerve action potential by phenytoin. Neuroscience Letters. 66: 163-8. PMID 3725182 DOI: 10.1016/0304-3940(86)90184-9 |
0.624 |
|
1986 |
Hevron E, David G, Arnon A, Yaari Y. Acetylcholine modulates two types of presynaptic potassium channels in vertebrate motor nerve terminals. Neuroscience Letters. 72: 87-92. PMID 2433646 DOI: 10.1016/0304-3940(86)90624-5 |
0.635 |
|
1985 |
Selzer ME, David G, Yaari Y. On the mechanism by which phenytoin blocks post-tetanic potentiation at the frog neuromuscular junction. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 5: 2894-9. PMID 4056860 DOI: 10.1523/Jneurosci.05-11-02894.1985 |
0.645 |
|
1985 |
Yaari Y, Selzer ME, David G. Frequency-dependent effects of phenytoin on frog junctional transmission: presynaptic mechanisms. Brain Research. 345: 102-10. PMID 2998545 DOI: 10.1016/0006-8993(85)90840-6 |
0.655 |
|
1985 |
David G, Selzer ME, Yaari Y. Suppression by phenytoin of convulsant-induced afterdischarges at presynaptic nerve terminals. Brain Research. 339: 57-65. PMID 2992698 DOI: 10.1016/0006-8993(85)90621-3 |
0.638 |
|
1984 |
Selzer ME, David G, Yaari Y. Phenytoin reduces frequency potentiation of synaptic potentials at the frog neuromuscular junction. Brain Research. 304: 149-52. PMID 6744034 DOI: 10.1016/0006-8993(84)90871-0 |
0.612 |
|
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