Robert C. Ritter - Publications

Affiliations: 
Washington State University, Pullman, WA, United States 
Area:
Ingestive behavior

106 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2015 Campos CA, Ritter RC. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 308: R1-9. PMID 25394828 DOI: 10.1152/ajpregu.00388.2014  1
2015 Zhao H, Peters JH, Zhu M, Page SJ, Ritter RC, Appleyard SM. Frequency-dependent facilitation of synaptic throughput via postsynaptic NMDA receptors in the nucleus of the solitary tract. The Journal of Physiology. 593: 111-25. PMID 25281729 DOI: 10.1113/jphysiol.2013.258103  1
2014 Campos CA, Shiina H, Ritter RC. Central vagal afferent endings mediate reduction of food intake by melanocortin-3/4 receptor agonist. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 12636-45. PMID 25232103 DOI: 10.1523/JNEUROSCI.1121-14.2014  1
2013 Campos CA, Shiina H, Silvas M, Page S, Ritter RC. Vagal afferent NMDA receptors modulate CCK-induced reduction of food intake through synapsin I phosphorylation in adult male rats. Endocrinology. 154: 2613-25. PMID 23715865 DOI: 10.1210/en.2013-1062  1
2012 Campos CA, Wright JS, Czaja K, Ritter RC. CCK-induced reduction of food intake and hindbrain MAPK signaling are mediated by NMDA receptor activation. Endocrinology. 153: 2633-46. PMID 22508518 DOI: 10.1210/en.2012-1025  1
2012 Gallaher ZR, Ryu V, Herzog T, Ritter RC, Czaja K. Changes in microglial activation within the hindbrain, nodose ganglia, and the spinal cord following subdiaphragmatic vagotomy. Neuroscience Letters. 513: 31-6. PMID 22342909 DOI: 10.1016/j.neulet.2012.01.079  1
2012 Zhang J, Ritter RC. Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 302: R264-73. PMID 22031786 DOI: 10.1152/ajpregu.00114.2011  1
2011 Wright J, Campos C, Herzog T, Covasa M, Czaja K, Ritter RC. Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 301: R448-55. PMID 21562094 DOI: 10.1152/ajpregu.00026.2011  1
2011 Ritter RC. A tale of two endings: modulation of satiation by NMDA receptors on or near central and peripheral vagal afferent terminals. Physiology & Behavior. 105: 94-9. PMID 21382391 DOI: 10.1016/j.physbeh.2011.02.042  1
2010 Ruiter M, Duffy P, Simasko S, Ritter RC. Increased hypothalamic signal transducer and activator of transcription 3 phosphorylation after hindbrain leptin injection. Endocrinology. 151: 1509-19. PMID 20185769 DOI: 10.1210/en.2009-0854  1
2009 Guard DB, Swartz TD, Ritter RC, Burns GA, Covasa M. NMDA NR2 receptors participate in CCK-induced reduction of food intake and hindbrain neuronal activation. Brain Research. 1266: 37-44. PMID 19232331 DOI: 10.1016/j.brainres.2009.02.003  1
2009 Guard DB, Swartz TD, Ritter RC, Burns GA, Covasa M. Blockade of hindbrain NMDA receptors containing NR2 subunits increases sucrose intake. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 296: R921-8. PMID 19193935 DOI: 10.1152/ajpregu.90456.2008  1
2008 Czaja K, Burns GA, Ritter RC. Capsaicin-induced neuronal death and proliferation of the primary sensory neurons located in the nodose ganglia of adult rats. Neuroscience. 154: 621-30. PMID 18456414 DOI: 10.1016/j.neuroscience.2008.03.055  1
2007 Peters JH, Simasko SM, Ritter RC. Leptin analog antagonizes leptin effects on food intake and body weight but mimics leptin-induced vagal afferent activation. Endocrinology. 148: 2878-85. PMID 17363463 DOI: 10.1210/en.2006-1320  1
2006 Czaja K, Ritter RC, Burns GA. Vagal afferent neurons projecting to the stomach and small intestine exhibit multiple N-methyl-D-aspartate receptor subunit phenotypes. Brain Research. 1119: 86-93. PMID 16989781 DOI: 10.1016/j.brainres.2006.08.042  1
2006 Peters JH, Simasko SM, Ritter RC. Modulation of vagal afferent excitation and reduction of food intake by leptin and cholecystokinin. Physiology & Behavior. 89: 477-85. PMID 16872644 DOI: 10.1016/j.physbeh.2006.06.017  1
2006 Czaja K, Ritter RC, Burns GA. N-methyl-D-aspartate receptor subunit phenotypes of vagal afferent neurons in nodose ganglia of the rat. The Journal of Comparative Neurology. 496: 877-85. PMID 16628619 DOI: 10.1002/cne.20955  1
2006 Peters JH, Ritter RC, Simasko SM. Leptin and CCK selectively activate vagal afferent neurons innervating the stomach and duodenum. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 290: R1544-9. PMID 16384857 DOI: 10.1152/ajpregu.00811.2005  1
2006 Hung CY, Covasa M, Ritter RC, Burns GA. Hindbrain administration of NMDA receptor antagonist AP-5 increases food intake in the rat. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 290: R642-51. PMID 16269572 DOI: 10.1152/ajpregu.00641.2005  1
2006 Peters JH, Ritter RC, Simasko SM. Leptin and CCK modulate complementary background conductances to depolarize cultured nodose neurons. American Journal of Physiology. Cell Physiology. 290: C427-32. PMID 16192299 DOI: 10.1152/ajpcell.00439.2005  1
2005 Gulley S, Covasa M, Ritter RC, Sayegh AI. Cholecystokinin1 receptors mediate the increase in Fos-like immunoreactivity in the rat myenteric plexus following intestinal oleate infusion. Physiology & Behavior. 86: 128-35. PMID 16098547 DOI: 10.1016/j.physbeh.2005.07.001  1
2005 Gillespie BR, Burns GA, Ritter RC. NMDA channels control meal size via central vagal afferent terminals. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 289: R1504-11. PMID 16020524 DOI: 10.1152/ajpregu.00169.2005  1
2005 Covasa M, Ritter RC. Reduced CCK-induced Fos expression in the hindbrain, nodose ganglia, and enteric neurons of rats lacking CCK-1 receptors. Brain Research. 1051: 155-63. PMID 16005445 DOI: 10.1016/j.brainres.2005.06.003  1
2005 van de Wall EH, Duffy P, Ritter RC. CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 289: R695-703. PMID 15905220 DOI: 10.1152/ajpregu.00809.2004  1
2005 Peters JH, McKay BM, Simasko SM, Ritter RC. Leptin-induced satiation mediated by abdominal vagal afferents. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 288: R879-84. PMID 15591156 DOI: 10.1152/ajpregu.00716.2004  1
2004 Sayegh AI, Covasa M, Ritter RC. Intestinal infusions of oleate and glucose activate distinct enteric neurons in the rat. Autonomic Neuroscience : Basic & Clinical. 115: 54-63. PMID 15507406 DOI: 10.1016/j.autneu.2004.08.006  1
2004 Covasa M, Hung CY, Ritter RC, Burns GA. Intracerebroventricular administration of MK-801 increases food intake through mechanisms independent of gastric emptying. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 287: R1462-7. PMID 15358605 DOI: 10.1152/ajpregu.00471.2004  1
2004 Ritter RC. Gastrointestinal mechanisms of satiation for food. Physiology & Behavior. 81: 249-73. PMID 15159171 DOI: 10.1016/j.physbeh.2004.02.012  1
2004 Ritter RC. Increased food intake and CCK receptor antagonists: beyond abdominal vagal afferents. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 286: R991-3. PMID 15142854 DOI: 10.1152/ajpregu.00116.2004  1
2004 Peters JH, Karpiel AB, Ritter RC, Simasko SM. Cooperative activation of cultured vagal afferent neurons by leptin and cholecystokinin. Endocrinology. 145: 3652-7. PMID 15105382 DOI: 10.1210/en.2004-0221  1
2004 Covasa M, Ritter RC, Burns GA. NMDA receptor blockade attenuates CCK-induced reduction of real feeding but not sham feeding. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 286: R826-31. PMID 14726428 DOI: 10.1152/ajpregu.00570.2003  1
2003 Simasko SM, Ritter RC. Cholecystokinin activates both A- and C-type vagal afferent neurons. American Journal of Physiology. Gastrointestinal and Liver Physiology. 285: G1204-13. PMID 12946940 DOI: 10.1152/ajpgi.00132.2003  1
2003 Covasa M, Ritter RC, Burns GA. Cholinergic neurotransmission participates in increased food intake induced by NMDA receptor blockade. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 285: R641-8. PMID 12775553 DOI: 10.1152/ajpregu.00055.2003  1
2003 Sayegh AI, Ritter RC. Cholecystokinin activates specific enteric neurons in the rat small intestine. Peptides. 24: 237-44. PMID 12668208 DOI: 10.1016/S0196-9781(03)00035-4  1
2003 Sayegh AI, Ritter RC. Morphology and distribution of nitric oxide synthase-, neurokinin-1 receptor-, calretinin-, calbindin-, and neurofilament-M-immunoreactive neurons in the myenteric and submucosal plexuses of the rat small intestine. The Anatomical Record. Part a, Discoveries in Molecular, Cellular, and Evolutionary Biology. 271: 209-16. PMID 12552637 DOI: 10.1002/ar.a.10024  1
2002 Simasko SM, Wiens J, Karpiel A, Covasa M, Ritter RC. Cholecystokinin increases cytosolic calcium in a subpopulation of cultured vagal afferent neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 283: R1303-13. PMID 12388458 DOI: 10.1152/ajpregu.00050.2002  1
2002 Matson CA, Reid DF, Ritter RC. Daily CCK injection enhances reduction of body weight by chronic intracerebroventricular leptin infusion. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 282: R1368-73. PMID 11959678 DOI: 10.1152/ajpregu.00080.2001  1
2001 Covasa M, Ritter RC. Attenuated satiation response to intestinal nutrients in rats that do not express CCK-A receptors. Peptides. 22: 1339-48. PMID 11457530 DOI: 10.1016/S0196-9781(01)00461-2  1
2001 Weller A, Gispan IH, Ritter RC, Brenner L, Smith GP. Release of endogenous cholecystokinin in response to gastric preloads in rats on postnatal days 9--12. Physiology & Behavior. 72: 1-4. PMID 11239974 DOI: 10.1016/S0031-9384(00)00288-2  1
2001 Covasa M, Marcuson JK, Ritter RC. Diminished satiation in rats exposed to elevated levels of endogenous or exogenous cholecystokinin. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 280: R331-7. PMID 11208559  1
2000 Covasa M, Grahn J, Ritter RC. Reduced hindbrain and enteric neuronal response to intestinal oleate in rats maintained on high-fat diet. Autonomic Neuroscience : Basic & Clinical. 84: 8-18. PMID 11109985 DOI: 10.1016/S1566-0702(00)00176-4  1
2000 Sayegh AI, Ritter RC. Vagus nerve participates in CCK-induced Fos expression in hindbrain but not myenteric plexus. Brain Research. 878: 155-62. PMID 10996146 DOI: 10.1016/S0006-8993(00)02731-1  1
2000 Treece BR, Ritter RC, Burns GA. Lesions of the dorsal vagal complex abolish increases in meal size induced by NMDA receptor blockade. Brain Research. 872: 37-43. PMID 10924673 DOI: 10.1016/S0006-8993(00)02432-X  1
2000 Covasa M, Ritter RC, Burns GA. NMDA receptor participation in control of food intake by the stomach. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 278: R1362-8. PMID 10801308  1
2000 Matson CA, Reid DF, Cannon TA, Ritter RC. Cholecystokinin and leptin act synergistically to reduce body weight. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 278: R882-90. PMID 10749775  1
2000 Sayegh AI, Ritter RC. CCK-A receptor activation induces fos expression in myenteric neurons of rat small intestine. Regulatory Peptides. 88: 75-81. PMID 10706955 DOI: 10.1016/S0167-0115(99)00124-X  1
2000 Covasa M, Grahn J, Ritter RC. High fat maintenance diet attenuates hindbrain neuronal response to CCK. Regulatory Peptides. 86: 83-8. PMID 10672906 DOI: 10.1016/S0167-0115(99)00084-1  1
2000 Covasa M, Ritter RC, Burns GA. Reduction of food intake by intestinal macronutrient infusion is not reversed by NMDA receptor blockade. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 278: R345-51. PMID 10666134  1
2000 Covasa M, Ritter RC. Adaptation to high-fat diet reduces inhibition of gastric emptying by CCK and intestinal oleate. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 278: R166-70. PMID 10644635  1
1999 Matson CA, Ritter RC. Long-term CCK-leptin synergy suggests a role for CCK in the regulation of body weight. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 276: R1038-R1045. PMID 29592497 DOI: 10.1152/ajpregu.1999.276.4.R1038  1
1999 Ritter RC, Covasa M, Matson CA. Cholecystokinin: proofs and prospects for involvement in control of food intake and body weight. Neuropeptides. 33: 387-99. PMID 10657516 DOI: 10.1054/npep.1999.0051  1
1999 Covasa M, Ritter RC. Reduced sensitivity to the satiation effect of intestinal oleate in rats adapted to high-fat diet. The American Journal of Physiology. 277: R279-85. PMID 10409283  1
1999 Matson CA, Ritter RC. Long-term CCK-leptin synergy suggests a role for CCK in the regulation of body weight. The American Journal of Physiology. 276: R1038-45. PMID 10198383  1
1999 Sayegh AI, Anderson NV, Harding JW, Cerpovicz P, DeBowes RM, Ritter RC, Baker GJ, Reeck G. Purification of two equine pepsinogens by use of high-performance liquid chromatography. American Journal of Veterinary Research. 60: 114-8. PMID 9918158  1
1998 Burns GA, Ritter RC. Visceral afferent participation in delayed satiation following NMDA receptor blockade. Physiology & Behavior. 65: 361-6. PMID 9855488 DOI: 10.1016/S0031-9384(98)00176-0  1
1998 Treece BR, Covasa M, Ritter RC, Burns GA. Delay in meal termination follows blockade of N-methyl-D-aspartate receptors in the dorsal hindbrain. Brain Research. 810: 34-40. PMID 9813231 DOI: 10.1016/S0006-8993(98)00867-1  1
1998 Covasa M, Ritter RC. Rats maintained on high-fat diets exhibit reduced satiety in response to CCK and bombesin. Peptides. 19: 1407-15. PMID 9809656 DOI: 10.1016/S0196-9781(98)00096-5  1
1998 Brenner LA, Ritter RC. Intracerebroventricular cholecystokinin A-receptor antagonist does not reduce satiation by endogenous CCK. Physiology & Behavior. 63: 711-6. PMID 9523920 DOI: 10.1016/S0031-9384(97)00519-2  1
1998 Burns GA, Fleischmann LG, Ritter RC. MK-801 interferes with nutrient-related signals for satiation. Appetite. 30: 1-12. PMID 9515046 DOI: 10.1006/appe.1997.0139  1
1997 Tamura CS, Ritter RC. Intraintestinal capsaicin transiently reduces CGRP-like immunoreactivity in rat submucosal plexus. Brain Research. 770: 248-55. PMID 9372226 DOI: 10.1016/S0006-8993(97)00855-X  1
1997 Weller A, Gispan IH, Armony-Sivan R, Ritter RC, Smith GP. Preloads of corn oil inhibit independent ingestion on postnatal day 15 in rats. Physiology & Behavior. 62: 871-4. PMID 9284510 DOI: 10.1016/S0031-9384(97)00256-4  1
1997 Ritter RC, Costa M, Brookes SH. Nuclear Fos immunoreactivity in guinea pig myenteric neurons following activation of motor activity. The American Journal of Physiology. 273: G498-507. PMID 9277431  1
1997 Burns GA, Ritter RC. The non-competitive NMDA antagonist MK-801 increases food intake in rats. Pharmacology, Biochemistry, and Behavior. 56: 145-9. PMID 8981622 DOI: 10.1016/S0091-3057(96)00171-2  1
1996 Brenner LA, Ritter RC. Type A CCK receptors mediate satiety effects of intestinal nutrients. Pharmacology, Biochemistry, and Behavior. 54: 625-31. PMID 8743639 DOI: 10.1016/0091-3057(95)02210-4  1
1995 Brenner L, Ritter RC. Peptide cholesystokinin receptor antagonist increases food intake in rats. Appetite. 24: 1-9. PMID 7537951 DOI: 10.1016/S0195-6663(95)80001-8  1
1994 Ritter RC, Brenner LA, Tamura CS. Endogenous CCK and the peripheral neural substrates of intestinal satiety. Annals of the New York Academy of Sciences. 713: 255-67. PMID 8185167 DOI: 10.1111/j.1749-6632.1994.tb44073.x  1
1994 Tamura CS, Ritter RC. Intestinal capsaicin transiently attenuates suppression of sham feeding by oleate. The American Journal of Physiology. 267: R561-8. PMID 8067469  1
1994 Kirk CA, Beverly JL, Ritter RC, Strieker MJ, Brenner L, Morris JG, Rogers QR. Diet-induced cholecystokinin release in cats. The Journal of Nutrition. 124: 2670S-2671S. PMID 7996264  1
1993 Brenner L, Yox DP, Ritter RC. Suppression of sham feeding by intraintestinal nutrients is not correlated with plasma cholecystokinin elevation. The American Journal of Physiology. 264: R972-6. PMID 8498606  1
1993 Ladenheim EE, Ritter RC. Caudal hindbrain participation in the suppression of feeding by central and peripheral bombesin. The American Journal of Physiology. 264: R1229-34. PMID 8322979  1
1992 Weller A, Corp ES, Tyrka A, Ritter RC, Brenner L, Gibbs J, Smith GP. Trypsin inhibitor and maternal reunion increase plasma cholecystokinin in neonatal rats. Peptides. 13: 939-41. PMID 1480517 DOI: 10.1016/0196-9781(92)90052-5  1
1992 Calingasan N, Ritter S, Ritter R, Brenner L. Low-dose near-celiac arterial cholecystokinin suppresses food intake in rats. The American Journal of Physiology. 263: R572-7. PMID 1415643  1
1992 Yox DP, Brenner L, Ritter RC. CCK-receptor antagonists attenuate suppression of sham feeding by intestinal nutrients. The American Journal of Physiology. 262: R554-61. PMID 1373576  1
1991 Yox DP, Stokesberry H, Ritter RC. Fourth ventricular capsaicin attenuates suppression of sham feeding induced by intestinal nutrients. The American Journal of Physiology. 260: R681-7. PMID 2012240  1
1991 Yox DP, Stokesberry H, Ritter RC. Vagotomy attenuates suppression of sham feeding induced by intestinal nutrients. The American Journal of Physiology. 260: R503-8. PMID 2000999  1
1991 Ladenheim EE, Ritter RC. Capsaicin attenuates bombesin-induced suppression of food intake. The American Journal of Physiology. 260: R263-6. PMID 1996711  1
1989 Edwards GL, Ritter RC. Lateral parabrachial lesions attenuate ingestive effects of area postrema lesions. The American Journal of Physiology. 256: R306-12. PMID 2916693  1
1989 Ritter RC, Ritter S, Ewart WR, Wingate DL. Capsaicin attenuates hindbrain neuron responses to circulating cholecystokinin. The American Journal of Physiology. 257: R1162-8. PMID 2589542  1
1988 South EH, Ritter RC. Capsaicin application to central or peripheral vagal fibers attenuates CCK satiety. Peptides. 9: 601-12. PMID 3420015 DOI: 10.1016/0196-9781(88)90171-4  1
1988 Ladenheim EE, Speth RC, Ritter RC. Reduction of CCK-8 binding in the nucleus of the solitary tract in unilaterally nodosectomized rats. Brain Research. 474: 125-9. PMID 3214705 DOI: 10.1016/0006-8993(88)90675-0  1
1988 Ladenheim EE, Ritter RC. Low-dose fourth ventricular bombesin selectively suppresses food intake. The American Journal of Physiology. 255: R988-93. PMID 3202231  1
1988 Yox DP, Ritter RC. Capsaicin attenuates suppression of sham feeding induced by intestinal nutrients. The American Journal of Physiology. 255: R569-74. PMID 3140678  1
1987 Dey MS, Krieger RI, Ritter RC. Paraquat-induced, dose-dependent conditioned taste aversions and weight loss mediated by the area postrema. Toxicology and Applied Pharmacology. 87: 212-21. PMID 3824381 DOI: 10.1016/0041-008X(87)90283-3  1
1986 Edwards GL, Ladenheim EE, Ritter RC. Dorsomedial hindbrain participation in cholecystokinin-induced satiety. The American Journal of Physiology. 251: R971-7. PMID 3777222  1
1986 Ritter RC, Kalivas P, Bernier S. Cholecystokinin-induced suppression of locomotion is attenuated in capsaicin pretreated rats. Peptides. 7: 587-90. PMID 3763436 DOI: 10.1016/0196-9781(86)90031-8  1
1986 South EH, Ritter RC. Substance P-containing trigeminal sensory neurons project to the nucleus of the solitary tract. Brain Research. 372: 283-9. PMID 3518865 DOI: 10.1016/0006-8993(86)91136-4  1
1986 Edwards GL, Ritter RC. Area postrema lesions: cause of overingestion is not altered visceral nerve function. The American Journal of Physiology. 251: R575-81. PMID 3463217  1
1985 Ritter RC, Ladenheim EE. Capsaicin pretreatment attenuates suppression of food intake by cholecystokinin. The American Journal of Physiology. 248: R501-4. PMID 3985191  1
1984 Ritter RC, Edwards GL. Area postrema lesions cause overconsumption of palatable foods but not calories. Physiology & Behavior. 32: 923-7. PMID 6494309 DOI: 10.1016/0031-9384(84)90279-8  1
1984 Ritter RC. The role of the rat in research on control of ingestion: a commentary and review of recent advances in understanding brain control of food intake. Journal of Animal Science. 59: 1373-80. PMID 6392277  1
1983 South EH, Ritter RC. Overconsumption of preferred foods following capsaicin pretreatment of the area postrema and adjacent nucleus of the solitary tract. Brain Research. 288: 243-51. PMID 6661619 DOI: 10.1016/0006-8993(83)90100-2  1
1983 Nonavinakere VK, Ritter RC. Feeding elicited by 2-deoxyglucose occurs in the absence of reduced glucose oxidation. Appetite. 4: 177-85. PMID 6421229  1
1982 Edwards GL, Ritter RC. Area postrema lesions increase drinking to angiotensin and extracellular dehydration. Physiology & Behavior. 29: 943-7. PMID 7156231 DOI: 10.1016/0031-9384(82)90348-1  1
1981 Edwards GL, Ritter RC. Ablation of the area postrema causes exaggerated consumption of preferred foods in the rat. Brain Research. 216: 265-76. PMID 7248778 DOI: 10.1016/0006-8993(81)90129-3  1
1981 Ritter RC, Slusser PG, Stone S. Glucoreceptors controlling feeding and blood glucose: location in the hindbrain. Science (New York, N.Y.). 213: 451-2. PMID 6264602  1
1980 Engeset RM, Ritter RC. Intracerebroventricular 2-DG causes feeding in the absence of other signs of glucoprivation. Brain Research. 202: 229-33. PMID 7427740 DOI: 10.1016/S0006-8993(80)80051-5  1
1980 Weick BG, Ritter S, Ritter RC. Plasma catecholamines: exaggerated elevation is associated with stress susceptibility. Physiology & Behavior. 24: 869-74. PMID 7403288 DOI: 10.1016/0031-9384(80)90143-2  1
1980 Slusser PG, Ritter RC. Increased feeding and hyperglycemia elicited by intracerebroventricular 5-thioglucose. Brain Research. 202: 474-8. PMID 6254614 DOI: 10.1016/0006-8993(80)90158-4  1
1978 Ritter RC, Roelke M, Neville M. Glucoprivic feeding behavior in absence of other signs of glucoprivation. The American Journal of Physiology. 234: E617-21. PMID 665765  1
1978 Ritter RC, Balch OK. Feeding in response to insulin but not to 2-deoxy-D-glucose in the hamster. The American Journal of Physiology. 234: E20-4. PMID 623243  1
1978 Ritter S, Pelzer NL, Ritter RC. Absence of glucoprivic feeding after stress suggest impairment of noradrenergic neuron function. Brain Research. 149: 399-411. PMID 566607 DOI: 10.1016/0006-8993(78)90483-3  1
1977 Ritter S, Ritter RC. Protection against stress-induced brain norepinephrine depletion after repeated 2-deoxy-D-glucose administration. Brain Research. 127: 179-84. PMID 861752 DOI: 10.1016/0006-8993(77)90391-2  1
1976 Ritter RC, Balch OK. Occurrence of insulin induced feeding but absence of 2 deoxy D glucose induced feeding in the hamster Physiologist. 19: 340.  1
1976 Ritter RC, Neville M. Hypothalamic noradrenaline turnover is increased during glucoprivic feeding Federation Proceedings. 35: No. 2406.  1
1975 Ritter RC, Epstein AN. Control of meal size by central noradrenergic action. Proceedings of the National Academy of Sciences of the United States of America. 72: 3740-3. PMID 1059162  1
1974 Ritter RC, Epstein AN. Saliva lost by grooming: a major item in the rat's water economy. Behavioral Biology. 11: 581-5. PMID 4415674 DOI: 10.1016/S0091-6773(74)90935-3  1
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