| Year |
Citation |
Score |
| 2019 |
Nandedkar-Kulkarni N, Vartak AR, Sucheck SJ, Wall KA, Quinn A, Morran MP, McInerney MF. Development of a Bioconjugate Platform for Modifying the Immune Response of Autoreactive Cytotoxic T Lymphocytes Involved in Type 1 Diabetes. Bioconjugate Chemistry. PMID 31274300 DOI: 10.1021/Acs.Bioconjchem.9B00332 |
0.545 |
|
| 2016 |
Marrero I, Aguilera C, Hamm DE, Quinn A, Kumar V. High-throughput sequencing reveals restricted TCR Vβ usage and public TCRβ clonotypes among pancreatic lymph node memory CD4(+) T cells and their involvement in autoimmune diabetes. Molecular Immunology. 74: 82-95. PMID 27161799 DOI: 10.1016/J.Molimm.2016.04.013 |
0.502 |
|
| 2015 |
Clark MP, Leaman DW, Hazelhurst LA, Hwang ES, Quinn A. An aza-anthrapyrazole negatively regulates Th1 activity and suppresses experimental autoimmune encephalomyelitis. International Immunopharmacology. 31: 74-87. PMID 26709219 DOI: 10.1016/J.Intimp.2015.12.014 |
0.469 |
|
| 2011 |
Kiraly A, Koffman B, Hacker M, Gunning W, Rasche S, Quinn A. A novel aza-anthrapyrazole blocks the progression of experimental autoimmune encephalomyelitis after the priming of autoimmunity. Clinical Immunology (Orlando, Fla.). 141: 304-16. PMID 21964417 DOI: 10.1016/J.Clim.2011.08.009 |
0.743 |
|
| 2011 |
Rasche SS, Phillips M, McInerney MF, Sercarz EE, Quinn A. IL-13Rα1 expression on β-cell-specific T cells in NOD mice. Diabetes. 60: 1716-25. PMID 21617187 DOI: 10.2337/Db10-1229 |
0.741 |
|
| 2010 |
Linkes S, Fry C, Quinn A. Antigen-Experienced CD4lo T Cells Are Linked to Deficient Contraction of the Immune Response in Autoimmune Diabetes. Autoimmune Diseases. 2010: 920148. PMID 21188239 DOI: 10.4061/2010/920148 |
0.581 |
|
| 2009 |
Quinn A. Are T-cell responses to GAD65 influential in type 1 diabetes? Diabetes. 58: 2729-30. PMID 19940237 DOI: 10.2337/Db09-1445 |
0.554 |
|
| 2009 |
Rasche S, Busick RY, Quinn A. GAD65-Specific Cytotoxic T Lymphocytes Mediate Beta-Cell Death and Loss of Function. The Review of Diabetic Studies : Rds. 6: 43-53. PMID 19557295 DOI: 10.1900/RDS.2009.6.43 |
0.7 |
|
| 2008 |
Dai YD, Jensen KP, Marrero I, Li N, Quinn A, Sercarz EE. N-terminal flanking residues of a diabetes-associated GAD65 determinant are necessary for activation of antigen-specific T cells in diabetes-resistant mice. European Journal of Immunology. 38: 968-76. PMID 18395850 DOI: 10.1002/Eji.200737703 |
0.635 |
|
| 2007 |
Busick RY, Aguilera C, Quinn A. Dominant CTL-inducing epitopes on GAD65 are adjacent to or overlap with dominant Th-inducing epitopes. Clinical Immunology (Orlando, Fla.). 122: 298-311. PMID 17174605 DOI: 10.1016/J.Clim.2006.10.008 |
0.745 |
|
| 2007 |
Mayo S, Quinn A. Altered susceptibility to EAE in congenic NOD mice: Altered processing of the encephalitogenic MOG35-55 peptide by NOR/LtJ mice. Clinical Immunology (Orlando, Fla.). 122: 91-100. PMID 16990050 DOI: 10.1016/J.Clim.2006.08.001 |
0.774 |
|
| 2006 |
Quinn A, McInerney M, Huffman D, McInerney B, Mayo S, Haskins K, Sercarz E. T cells to a dominant epitope of GAD65 express a public CDR3 motif. International Immunology. 18: 967-79. PMID 16641112 DOI: 10.1093/Intimm/Dxl033 |
0.789 |
|
| 2006 |
Mayo S, Kohler W, Kumar V, Quinn A. Insulin-dependent diabetes loci Idd5 and Idd9 increase sensitivity to experimental autoimmune encephalomyelitis. Clinical Immunology (Orlando, Fla.). 118: 219-28. PMID 16301001 DOI: 10.1016/J.Clim.2005.10.006 |
0.758 |
|
| 2004 |
Im JS, Quinn A, Sercarz EE, Lake DF. Molecular profile of the T cell receptors of regulatory and effector CD4+ T cells recognizing overlapping determinants on glutamic acid decarboxylase (524-543). Molecular Immunology. 40: 971-80. PMID 14725793 DOI: 10.1016/J.Molimm.2003.09.007 |
0.499 |
|
| 2003 |
Quinn A. Antigen-induced T1D in NOD mice. Journal of Autoimmunity. 20: 207-10. PMID 12753805 DOI: 10.1016/S0896-8411(03)00028-3 |
0.597 |
|
| 2002 |
Melo ME, Gabaglia CR, Moudgil KD, Sercarz EE, Quinn A. Strain-dependent effect of nasal instillation of antigen on the immune response in mice. The Israel Medical Association Journal : Imaj. 4: 902-7. PMID 12455176 |
0.49 |
|
| 2002 |
Quinn A, Melo M, Ethell D, Sercarz EE. Relative resistance to nasally induced tolerance in non-obese diabetic mice but not other I-A(g7)-expressing mouse strains. International Immunology. 13: 1321-33. PMID 11581177 DOI: 10.1093/Intimm/13.10.1321 |
0.588 |
|
| 2002 |
Quinn A, Kumar V, Jensen KP, Sercarz EE. Interactions of effectors and regulators are decisive in the manifestations of type 1 diabetes in nonobese diabetic mice. Current Directions in Autoimmunity. 4: 171-92. PMID 11569402 DOI: 10.1159/000060537 |
0.444 |
|
| 2001 |
Kaufman DL, Tisch R, Sarvetnick N, Chatenoud L, Harrison LC, Haskins K, Quinn A, Sercarz E, Singh B, Von Herrath M, Wegmann D, Wen L, Zekzer D. Report from the 1st International NOD Mouse T-Cell Workshop and the Follow-Up Mini-Workshop Diabetes. 50: 2459-2463. PMID 11679422 DOI: 10.2337/Diabetes.50.11.2459 |
0.566 |
|
| 2001 |
Quinn A, McInerney MF, Sercarz EE. MHC class I-restricted determinants on the glutamic acid decarboxylase 65 molecule induce spontaneous CTL activity. Journal of Immunology (Baltimore, Md. : 1950). 167: 1748-57. PMID 11466400 DOI: 10.4049/Jimmunol.167.3.1748 |
0.608 |
|
| 2001 |
Quinn A, McInerney B, Reich EP, Kim O, Jensen KP, Sercarz EE. Regulatory and effector CD4 T cells in nonobese diabetic mice recognize overlapping determinants on glutamic acid decarboxylase and use distinct V beta genes. Journal of Immunology (Baltimore, Md. : 1950). 166: 2982-91. PMID 11207247 DOI: 10.4049/Jimmunol.166.5.2982 |
0.653 |
|
| 2000 |
Maverakis E, Beech JT, Wilson SS, Quinn A, Pedersen B, Sercarz EE. T cell receptor complementarity determining region 3 length analysis reveals the absence of a characteristic public T cell repertoire in neonatal tolerance. The response in the "tolerant" mouse within the residual repertoire is quantitatively similar but qualitatively different. The Journal of Experimental Medicine. 191: 695-702. PMID 10684861 DOI: 10.1084/Jem.191.4.695 |
0.572 |
|
| 1996 |
Quinn A, Sercarz EE. T cells with multiple fine specificities are used by non-obese diabetic (NOB) mice in the response to GAD(524-543) Journal of Autoimmunity. 9: 365-370. PMID 8816972 DOI: 10.1006/Jaut.1996.0049 |
0.513 |
|
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