Year |
Citation |
Score |
2013 |
Melzer N, Hicking G, Bittner S, Bobak N, Göbel K, Herrmann AM, Wiendl H, Meuth SG. Excitotoxic neuronal cell death during an oligodendrocyte-directed CD8+ T cell attack in the CNS gray matter. Journal of Neuroinflammation. 10: 121. PMID 24093512 DOI: 10.1186/1742-2094-10-121 |
0.3 |
|
2011 |
Melzer N, Meuth SG, Torres-Salazar D, Bittner S, Zozulya AL, Weidenfeller C, Kotsiari A, Stangel M, Fahlke C, Wiendl H. Correction: A β-Lactam Antibiotic Dampens Excitotoxic Inflammatory CNS Damage in a Mouse Model of Multiple Sclerosis Plos One. 6. DOI: 10.1371/annotation/b1ff1c14-fa84-40a0-b095-e5ee47c74125 |
0.67 |
|
2009 |
Meuth SG, Melzer N, Kleinschnitz C, Budde T, Wiendl H. [Multiple sclerosis -- a channelopathy? Targeting ion channels and transporters in inflammatory neurodegeneration]. Der Nervenarzt. 80: 422-9. PMID 19011824 DOI: 10.1007/s00115-008-2599-7 |
0.331 |
|
2008 |
Melzer N, Meuth SG, Torres-Salazar D, Bittner S, Zozulya AL, Weidenfeller C, Kotsiari A, Stangel M, Fahlke C, Wiendl H. A beta-lactam antibiotic dampens excitotoxic inflammatory CNS damage in a mouse model of multiple sclerosis. Plos One. 3: e3149. PMID 18773080 DOI: 10.1371/Journal.Pone.0003149 |
0.666 |
|
2008 |
Melzer N, Meuth SG, Torres-Salazar D, Bittner S, Zozulya AL, Weidenfeller C, Kotsiari A, Stangel M, Fahlke C, Wiendl H. Correction: A β-Lactam Antibiotic Dampens Excitotoxic Inflammatory CNS Damage in a Mouse Model of Multiple Sclerosis Plos One. 3. DOI: 10.1371/annotation/b898d6ee-801a-474e-9776-72e4da664b89 |
0.67 |
|
2005 |
Melzer N, Torres-Salazar D, Fahlke C. A dynamic switch between inhibitory and excitatory currents in a neuronal glutamate transporter. Proceedings of the National Academy of Sciences of the United States of America. 102: 19214-8. PMID 16365297 DOI: 10.1073/Pnas.0508837103 |
0.625 |
|
2003 |
Melzer N, Biela A, Fahlke C. Glutamate modifies ion conduction and voltage-dependent gating of excitatory amino acid transporter-associated anion channels. The Journal of Biological Chemistry. 278: 50112-9. PMID 14506254 DOI: 10.1074/Jbc.M307990200 |
0.611 |
|
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