Shaun M. Sheets, Ph.D. - Publications
Affiliations: | 2006 | Loma Linda University, Loma Linda, United States |
Area:
Microbiology Biology, Cell BiologyYear | Citation | Score | |||
---|---|---|---|---|---|
2008 | Sheets SM, Robles-Price AG, McKenzie RM, Casiano CA, Fletcher HM. Gingipain-dependent interactions with the host are important for survival of Porphyromonas gingivalis. Frontiers in Bioscience : a Journal and Virtual Library. 13: 3215-38. PMID 18508429 DOI: 10.2741/2922 | 0.637 | |||
2006 | Vanterpool E, Roy F, Zhan W, Sheets SM, Sangberg L, Fletcher HM. VimA is part of the maturation pathway for the major gingipains of Porphyromonas gingivalis W83. Microbiology (Reading, England). 152: 3383-9. PMID 17074907 DOI: 10.1099/Mic.0.29146-0 | 0.571 | |||
2006 | Sheets SM, Potempa J, Travis J, Fletcher HM, Casiano CA. Gingipains from Porphyromonas gingivalis W83 synergistically disrupt endothelial cell adhesion and can induce caspase-independent apoptosis. Infection and Immunity. 74: 5667-78. PMID 16988242 DOI: 10.1128/Iai.01140-05 | 0.626 | |||
2005 | Sheets SM, Potempa J, Travis J, Casiano CA, Fletcher HM. Gingipains from Porphyromonas gingivalis W83 induce cell adhesion molecule cleavage and apoptosis in endothelial cells. Infection and Immunity. 73: 1543-52. PMID 15731052 DOI: 10.1128/Iai.73.3.1543-1552.2005 | 0.612 | |||
2003 | Olango GJ, Roy F, Sheets SM, Young MK, Fletcher HM. Gingipain RgpB is excreted as a proenzyme in the vimA-defective mutant Porphyromonas gingivalis FLL92. Infection and Immunity. 71: 3740-7. PMID 12819055 DOI: 10.1128/Iai.71.7.3740-3747.2003 | 0.526 | |||
Show low-probability matches. |