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Amy J. Hawkins, Ph.D. - Publications

Affiliations: 
2009 Human Genetics Virginia Commonwealth University, Richmond, VA, United States 
Area:
Molecular Biology, Oncology, Genetics
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6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2011 Hawkins AJ, Golding SE, Khalil A, Valerie K. DNA double-strand break - induced pro-survival signaling. Radiotherapy and Oncology : Journal of the European Society For Therapeutic Radiology and Oncology. 101: 13-7. PMID 21726915 DOI: 10.1016/J.Radonc.2011.05.074  0.418
2010 Shultz JC, Goehe RW, Wijesinghe DS, Murudkar C, Hawkins AJ, Shay JW, Minna JD, Chalfant CE. Alternative splicing of caspase 9 is modulated by the phosphoinositide 3-kinase/Akt pathway via phosphorylation of SRp30a. Cancer Research. 70: 9185-96. PMID 21045158 DOI: 10.1158/0008-5472.Can-10-1545  0.373
2010 Adams BR, Hawkins AJ, Povirk LF, Valerie K. ATM-independent, high-fidelity nonhomologous end joining predominates in human embryonic stem cells. Aging. 2: 582-96. PMID 20844317  0.567
2010 Hawkins AJ, White DE, Golding SE, Akopiants K, Rauscher FJ, Povirk LF, Valerie K. Abstract 3923: ATM and KAP1 are required for nonhomologous end-joining in transcriptionally active chromatin Cancer Research. 70: 3923-3923. DOI: 10.1158/1538-7445.Am10-3923  0.582
2009 Golding SE, Morgan RN, Adams BR, Hawkins AJ, Povirk LF, Valerie K. Pro-survival AKT and ERK signaling from EGFR and mutant EGFRvIII enhances DNA double-strand break repair in human glioma cells. Cancer Biology & Therapy. 8: 730-8. PMID 19252415 DOI: 10.4161/Cbt.8.8.7927  0.578
2009 Hawkins AJ, Subler MA, Akopiants K, Wiley JL, Taylor SM, Rice AC, Windle JJ, Valerie K, Povirk LF. In vitro complementation of Tdp1 deficiency indicates a stabilized enzyme-DNA adduct from tyrosyl but not glycolate lesions as a consequence of the SCAN1 mutation. Dna Repair. 8: 654-63. PMID 19211312 DOI: 10.1016/J.Dnarep.2008.12.012  0.55
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