Year |
Citation |
Score |
2023 |
Quiñonez M, DiFranco M, Wu F, Cannon SC. Retigabine suppresses loss of force in mouse models of hypokalaemic periodic paralysis. Brain : a Journal of Neurology. PMID 36718088 DOI: 10.1093/brain/awac441 |
0.633 |
|
2022 |
Wu F, Quiñonez M, Difranco M, Cannon S. CaV1.1 defects in hypokalemic periodic paralysis and harnessing multiple approaches for therapeutic intervention. The Journal of General Physiology. 154. PMID 34767012 DOI: 10.1085/jgp.2021ecc40 |
0.463 |
|
2021 |
Savalli N, Angelini M, Steccanella F, Wier J, Wu F, Quinonez M, DiFranco M, Neely A, Cannon SC, Olcese R. The distinct role of the four voltage sensors of the skeletal CaV1.1 channel in voltage-dependent activation. The Journal of General Physiology. 153. PMID 34546289 DOI: 10.1085/jgp.202112915 |
0.668 |
|
2021 |
Wu F, Quinonez M, Cannon SC. Gating pore currents occur in CaV1.1 domain III mutants associated with HypoPP. The Journal of General Physiology. 153. PMID 34463712 DOI: 10.1085/jgp.202112946 |
0.633 |
|
2020 |
Kubota T, Wu F, Vicart S, Nakaza M, Sternberg D, Watanabe D, Furuta M, Kokunai Y, Abe T, Kokubun N, Fontaine B, Cannon SC, Takahashi MP. Hypokalaemic periodic paralysis with a charge-retaining substitution in the voltage sensor. Brain Communications. 2: fcaa103. PMID 33005891 DOI: 10.1093/braincomms/fcaa103 |
0.74 |
|
2020 |
Savalli N, Angelini M, Steccanella F, Wu F, Quinonez M, Neely A, Cannon SC, Olcese R. The Contribution of the Individual Voltage Sensors to the Activation of Skeletal CaV1.1 Channels Biophysical Journal. 118: 105a. DOI: 10.1016/J.Bpj.2019.11.728 |
0.448 |
|
2019 |
Mason ER, Wu F, Patel RR, Xiao Y, Cannon SC, Cummins TR. Resurgent and Gating Pore Currents Induced by de novo Epilepsy Mutations. Eneuro. PMID 31558572 DOI: 10.1523/ENEURO.0141-19.2019 |
0.662 |
|
2019 |
Mi W, Wu F, Quinonez M, DiFranco M, Cannon SC. Recovery from acidosis is a robust trigger for loss of force in murine hypokalemic periodic paralysis. The Journal of General Physiology. PMID 30733232 DOI: 10.1085/Jgp.201812231 |
0.647 |
|
2019 |
Savalli N, Wu F, Quinonez M, Cannon SC, Olcese R. A Mutation Linked to Malignant Hyperthermia in the Skeletal CaV1.1 Channel Stabilizes the Resting State of Voltage Sensor I and Impairs Channel Activation Biophysical Journal. 116: 111a. DOI: 10.1016/J.Bpj.2018.11.629 |
0.683 |
|
2018 |
Wu F, Quinonez M, DiFranco M, Cannon SC. Stac3 enhances expression of human CaV1.1 in Xenopus oocytes and reveals gating pore currents in HypoPP mutant channels. The Journal of General Physiology. PMID 29386226 DOI: 10.1085/Jgp.201711962 |
0.73 |
|
2018 |
Wu F, Quinonez M, Cannon SC. Gating Pore Currents in DIII Hypopp Mutations of CaV1.1 Biophysical Journal. 114: 639a. DOI: 10.1016/J.Bpj.2017.11.3449 |
0.443 |
|
2018 |
Savalli N, Wu F, Quinonez M, Cannon SC, Olcese R. Optically-Tracked Structural Rearrangements of the Voltage Sensing Domains in the Human CaV1.1 Channel Biophysical Journal. 114: 39a. DOI: 10.1016/J.Bpj.2017.11.265 |
0.618 |
|
2017 |
Wu F, Cannon SC. Stac3 Facilitated Expression of Ca V 1.1 in Xenopus Oocytes to Assess Functional Consequences of Hypopp Mutant Ca V 1.1-R528H Biophysical Journal. 112: 245a. DOI: 10.1016/J.Bpj.2016.11.1341 |
0.327 |
|
2016 |
Wu F, Mi W, Fu Y, Struyk A, Cannon SC. Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis. Brain : a Journal of Neurology. PMID 27048647 DOI: 10.1093/Brain/Aww070 |
0.775 |
|
2016 |
Nelson BR, Makarewich CA, Anderson DM, Winders BR, Troupes CD, Wu F, Reese AL, McAnally JR, Chen X, Kavalali ET, Cannon SC, Houser SR, Bassel-Duby R, Olson EN. A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle. Science (New York, N.Y.). 351: 271-275. PMID 26816378 DOI: 10.1126/Science.Aad4076 |
0.639 |
|
2016 |
Cannon SC, Mi W, Wu F. Proposed Mechanism for Post‐Exercise Weakness in Hypokalemic Periodic Paralysis The Faseb Journal. 30. DOI: 10.1096/Fasebj.30.1_Supplement.1224.49 |
0.429 |
|
2016 |
Wu F, Mi W, Fu Y, Cannon SC. Mice with a Null Allele for NaV1.4 Exhibit Pseudo-Myasthenia, but are not Susceptibile to Periodic Paralysis Biophysical Journal. 110: 111a. DOI: 10.1016/J.Bpj.2015.11.656 |
0.613 |
|
2013 |
Wu F, Mi W, Cannon SC. Beneficial effects of bumetanide in a CaV1.1-R528H mouse model of hypokalaemic periodic paralysis. Brain : a Journal of Neurology. 136: 3766-74. PMID 24142145 DOI: 10.1093/Brain/Awt280 |
0.734 |
|
2013 |
Nelson BR, Wu F, Liu Y, Anderson DM, McAnally J, Lin W, Cannon SC, Bassel-Duby R, Olson EN. Skeletal muscle-specific T-tubule protein STAC3 mediates voltage-induced Ca2+ release and contractility. Proceedings of the National Academy of Sciences of the United States of America. 110: 11881-6. PMID 23818578 DOI: 10.1073/Pnas.1310571110 |
0.669 |
|
2013 |
Wu F, Mi W, Cannon SC. Bumetanide prevents transient decreases in muscle force in murine hypokalemic periodic paralysis. Neurology. 80: 1110-6. PMID 23427324 DOI: 10.1212/Wnl.0B013E3182886A0E |
0.685 |
|
2012 |
Wu F, Mi W, Hernández-Ochoa EO, Burns DK, Fu Y, Gray HF, Struyk AF, Schneider MF, Cannon SC. A calcium channel mutant mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 122: 4580-91. PMID 23187123 DOI: 10.1172/Jci66091 |
0.783 |
|
2012 |
Liu Y, Sugiura Y, Wu F, Mi W, Taketo MM, Cannon S, Carroll T, Lin W. β-Catenin stabilization in skeletal muscles, but not in motor neurons, leads to aberrant motor innervation of the muscle during neuromuscular development in mice. Developmental Biology. 366: 255-67. PMID 22537499 DOI: 10.1016/J.Ydbio.2012.04.003 |
0.622 |
|
2011 |
Wu F, Mi W, Burns DK, Fu Y, Gray HF, Struyk AF, Cannon SC. A sodium channel knockin mutant (NaV1.4-R669H) mouse model of hypokalemic periodic paralysis. The Journal of Clinical Investigation. 121: 4082-94. PMID 21881211 DOI: 10.1172/Jci57398 |
0.796 |
|
2009 |
Webb J, Wu FF, Cannon SC. Slow inactivation of the NaV1.4 sodium channel in mammalian cells is impeded by co-expression of the beta1 subunit. Pflã¼Gers Archiv : European Journal of Physiology. 457: 1253-63. PMID 18941776 DOI: 10.1007/S00424-008-0600-8 |
0.707 |
|
2008 |
Hayward LJ, Kim JS, Lee MY, Zhou H, Kim JW, Misra K, Salajegheh M, Wu FF, Matsuda C, Reid V, Cros D, Hoffman EP, Renaud JM, Cannon SC, Brown RH. Targeted mutation of mouse skeletal muscle sodium channel produces myotonia and potassium-sensitive weakness. The Journal of Clinical Investigation. 118: 1437-49. PMID 18317596 DOI: 10.1172/Jci32638 |
0.769 |
|
2005 |
Wu FF, Gordon E, Hoffman EP, Cannon SC. A C-terminal skeletal muscle sodium channel mutation associated with myotonia disrupts fast inactivation. The Journal of Physiology. 565: 371-80. PMID 15774523 DOI: 10.1113/Jphysiol.2005.082909 |
0.709 |
|
2002 |
Wu FF, Ryan A, Devaney J, Warnstedt M, Korade-Mirnics Z, Poser B, Escriva MJ, Pegoraro E, Yee AS, Felice KJ, Giuliani MJ, Mayer RF, Mongini T, Palmucci L, Marino M, et al. Novel CLCN1 mutations with unique clinical and electrophysiological consequences. Brain : a Journal of Neurology. 125: 2392-407. PMID 12390967 DOI: 10.1093/Brain/Awf246 |
0.459 |
|
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