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Pamela N. Lopert, Ph.D. - Publications

Affiliations: 
2014 Neuroscience University of Colorado Anschutz Medical Campus, Denver, Aurora, CO 
Area:
Neuroscience Biology, Biochemistry
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6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2024 Keeney JG, Astling D, Andries V, Vandepoele K, Anderson N, Davis JM, Lopert P, Vandenbussche J, Gevaert K, Staes A, Paukovich N, Vögeli B, Jones KL, van Roy F, Patel M, et al. Olduvai domain expression downregulates mitochondrial pathways: implications for human brain evolution and neoteny. Biorxiv : the Preprint Server For Biology. PMID 39484454 DOI: 10.1101/2024.10.21.619278  0.521
2015 Lopert P, Patel M. Mitochondrial mechanisms of redox cycling agents implicated in Parkinson's disease. Journal of Neural Transmission (Vienna, Austria : 1996). PMID 25749885 DOI: 10.1007/S00702-015-1386-4  0.564
2014 Shrotriya S, Deep G, Lopert P, Patel M, Agarwal R, Agarwal C. Grape seed extract targets mitochondrial electron transport chain complex III and induces oxidative and metabolic stress leading to cytoprotective autophagy and apoptotic death in human head and neck cancer cells. Molecular Carcinogenesis. PMID 25557495 DOI: 10.1002/Mc.22246  0.569
2014 Lopert P, Patel M. Brain mitochondria from DJ-1 knockout mice show increased respiration-dependent hydrogen peroxide consumption. Redox Biology. 2: 667-72. PMID 24936441 DOI: 10.1016/J.Redox.2014.04.010  0.582
2014 Lopert P, Patel M. Nicotinamide nucleotide transhydrogenase (Nnt) links the substrate requirement in brain mitochondria for hydrogen peroxide removal to the thioredoxin/peroxiredoxin (Trx/Prx) system. The Journal of Biological Chemistry. 289: 15611-20. PMID 24722990 DOI: 10.1074/Jbc.M113.533653  0.633
2012 Lopert P, Day BJ, Patel M. Thioredoxin reductase deficiency potentiates oxidative stress, mitochondrial dysfunction and cell death in dopaminergic cells. Plos One. 7: e50683. PMID 23226354 DOI: 10.1371/Journal.Pone.0050683  0.635
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