Year |
Citation |
Score |
2014 |
Kebede MA, Oler AT, Gregg T, Balloon AJ, Johnson A, Mitok K, Rabaglia M, Schueler K, Stapleton D, Thorstenson C, Wrighton L, Floyd BJ, Richards O, Raines S, Eliceiri K, et al. SORCS1 is necessary for normal insulin secretory granule biogenesis in metabolically stressed β cells. The Journal of Clinical Investigation. 124: 4240-56. PMID 25157818 DOI: 10.1172/Jci74072 |
0.747 |
|
2011 |
Raines SM, Richards OC, Schneider LR, Schueler KL, Rabaglia ME, Oler AT, Stapleton DS, Genové G, Dawson JA, Betsholtz C, Attie AD. Loss of PDGF-B activity increases hepatic vascular permeability and enhances insulin sensitivity. American Journal of Physiology. Endocrinology and Metabolism. 301: E517-26. PMID 21673305 DOI: 10.1152/Ajpendo.00241.2011 |
0.724 |
|
2010 |
Lane RF, Raines SM, Steele JW, Ehrlich ME, Lah JA, Small SA, Tanzi RE, Attie AD, Gandy S. Diabetes-associated SorCS1 regulates Alzheimer's amyloid-beta metabolism: evidence for involvement of SorL1 and the retromer complex. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 13110-5. PMID 20881129 DOI: 10.1523/Jneurosci.3872-10.2010 |
0.572 |
|
2010 |
Richards OC, Raines SM, Attie AD. The role of blood vessels, endothelial cells, and vascular pericytes in insulin secretion and peripheral insulin action. Endocrine Reviews. 31: 343-63. PMID 20164242 DOI: 10.1210/Er.2009-0035 |
0.722 |
|
2010 |
Lane RF, Raines SM, Levey A, Lah JJ, Small SA, Tanzi RE, Attie AD, Gandy SE. Diabetes-linked SORCS1, a gamma-secretase substrate and regulator of Alzheimer APP metabolism, forms complexes with multiple Alzheimer-linked proteins (APP, PS1, BACE, VPS35, and SORLA) Alzheimers & Dementia. 6. DOI: 10.1016/J.Jalz.2010.05.814 |
0.553 |
|
2006 |
Clee SM, Yandell BS, Schueler KM, Rabaglia ME, Richards OC, Raines SM, Kabara EA, Klass DM, Mui ET, Stapleton DS, Gray-Keller MP, Young MB, Stoehr JP, Lan H, Boronenkov I, et al. Positional cloning of Sorcs1, a type 2 diabetes quantitative trait locus. Nature Genetics. 38: 688-93. PMID 16682971 DOI: 10.1038/Ng1796 |
0.434 |
|
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