| Year |
Citation |
Score |
| 2013 |
Zhou Q, Pratt EB, Shyng SL. Engineered Kir6.2 mutations that correct the trafficking defect of K(ATP) channels caused by specific SUR1 mutations. Channels (Austin, Tex.). 7: 313-7. PMID 23695995 DOI: 10.4161/Chan.25003 |
0.746 |
|
| 2012 |
Pratt EB, Zhou Q, Gay JW, Shyng SL. Engineered interaction between SUR1 and Kir6.2 that enhances ATP sensitivity in KATP channels. The Journal of General Physiology. 140: 175-87. PMID 22802363 DOI: 10.1085/Jgp.201210803 |
0.814 |
|
| 2012 |
Shyng SL, Bushman JD, Pratt EB, Zhou Q. Molecular defects of ATP-sensitive potassium channels in congenital hyperinsulinism Monogenic Hyperinsulinemic Hypoglycemia Disorders. 21: 30-42. DOI: 10.1159/000334485 |
0.7 |
|
| 2011 |
Pratt EB, Shyng SL. ATP activates ATP-sensitive potassium channels composed of mutant sulfonylurea receptor 1 and Kir6.2 with diminished PIP2 sensitivity. Channels (Austin, Tex.). 5: 314-9. PMID 21654216 DOI: 10.4161/Chan.5.4.16510 |
0.805 |
|
| 2011 |
Pratt EB, Tewson P, Bruederle CE, Skach WR, Shyng SL. N-terminal transmembrane domain of SUR1 controls gating of Kir6.2 by modulating channel sensitivity to PIP2. The Journal of General Physiology. 137: 299-314. PMID 21321069 DOI: 10.1085/Jgp.201010557 |
0.803 |
|
| 2011 |
Pratt EB, Shyng SL. ATP activates ATP-sensitive potassium channels composed of mutant sulfonylurea receptor 1 and Kir6.2 with diminished PIP 2 sensitivity Channels. 5. |
0.771 |
|
| 2010 |
Yan FF, Pratt EB, Chen PC, Wang F, Skach WR, David LL, Shyng SL. Role of Hsp90 in biogenesis of the beta-cell ATP-sensitive potassium channel complex. Molecular Biology of the Cell. 21: 1945-54. PMID 20427569 DOI: 10.1091/Mbc.E10-02-0116 |
0.786 |
|
| 2010 |
Pratt EB, Tewson P, Shyng S. Sulfonylurea Receptor Transmembrane Domain Zero Mutations that Disrupt Full Length and Minimal ATP-Sensitive Potassium Channel Properties Biophysical Journal. 98: 699a. DOI: 10.1016/J.Bpj.2009.12.3838 |
0.784 |
|
| 2009 |
Fu Y, Tian W, Pratt EB, Dirling LB, Shyng SL, Meshul CK, Cohen DM. Down-regulation of ZnT8 expression in INS-1 rat pancreatic beta cells reduces insulin content and glucose-inducible insulin secretion. Plos One. 4: e5679. PMID 19479076 DOI: 10.1371/Journal.Pone.0005679 |
0.549 |
|
| 2009 |
Pratt EB, Yan FF, Gay JW, Stanley CA, Shyng SL. Sulfonylurea receptor 1 mutations that cause opposite insulin secretion defects with chemical chaperone exposure. The Journal of Biological Chemistry. 284: 7951-9. PMID 19151370 DOI: 10.1074/Jbc.M807012200 |
0.801 |
|
| 2006 |
Lin CW, Lin YW, Yan FF, Casey J, Kochhar M, Pratt EB, Shyng SL. Kir6.2 mutations associated with neonatal diabetes reduce expression of ATP-sensitive K+ channels: implications in disease mechanism and sulfonylurea therapy. Diabetes. 55: 1738-46. PMID 16731837 DOI: 10.2337/Db05-1571 |
0.796 |
|
| 2005 |
Pratt EB, Brink TS, Bergson P, Voigt MM, Cook SP. Use-dependent inhibition of P2X3 receptors by nanomolar agonist. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 25: 7359-65. PMID 16093386 DOI: 10.1523/Jneurosci.5189-04.2005 |
0.469 |
|
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