Aldrin E. Molero, Ph.D. - Publications

Affiliations: 
2008 Yeshiva University, New York, NY, United States 
Area:
Neuroscience Biology

11/20 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Mehler MF, Petronglo JR, Arteaga-Bracho EE, Gulinello M, Winchester ML, Pichamoorthy N, Young SK, DeJesus CD, Ishtiaq H, Gokhan S, Molero AE. Loss-of-huntingtin in medial and lateral ganglionic lineages differentially disrupts regional interneuron and projection neuron subtypes and promotes Huntington's disease-associated behavioral, cellular and pathological hallmarks. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 30626701 DOI: 10.1523/Jneurosci.2443-18.2018  0.76
2016 Arteaga-Bracho EE, Gulinello M, Winchester ML, Pichamoorthy N, Petronglo JR, Zambrano AD, Inocencio J, De Jesus CD, Louie JO, Gokhan S, Mehler MF, Molero AE. Postnatal and adult consequences of loss of huntingtin during development: Implications for Huntington's disease. Neurobiology of Disease. PMID 27623015 DOI: 10.1016/J.Nbd.2016.09.006  0.751
2016 Molero AE, Arteaga-Bracho EE, Chen CH, Gulinello M, Winchester ML, Pichamoorthy N, Gokhan S, Khodakhah K, Mehler MF. Selective expression of mutant huntingtin during development recapitulates characteristic features of Huntington's disease. Proceedings of the National Academy of Sciences of the United States of America. PMID 27140644 DOI: 10.1073/Pnas.1603871113  0.747
2014 Nguyen GD, Gokhan S, Molero AE, Yang SM, Kim BJ, Skoultchi AI, Mehler MF. The role of H1 linker histone subtypes in preserving the fidelity of elaboration of mesendodermal and neuroectodermal lineages during embryonic development. Plos One. 9: e96858. PMID 24802750 DOI: 10.1371/Journal.Pone.0096858  0.704
2013 Nguyen GD, Molero AE, Gokhan S, Mehler MF. Functions of huntingtin in germ layer specification and organogenesis. Plos One. 8: e72698. PMID 23967334 DOI: 10.1371/Journal.Pone.0072698  0.729
2013 Nguyen GD, Gokhan S, Molero AE, Mehler MF. Selective roles of normal and mutant huntingtin in neural induction and early neurogenesis. Plos One. 8: e64368. PMID 23691206 DOI: 10.1371/Journal.Pone.0064368  0.753
2013 Nguyen GD, Molero AE, Gokhan S, Mehler MF. Correction: Functions of Huntingtin in Germ Layer Specification and Organogenesis Plos One. 8. DOI: 10.1371/Annotation/Edee8Dfa-6B2A-44F4-866A-098F186E27F0  0.528
2013 Molero A, Mehler MF. Huntington's disease Neuroscience in the 21st Century: From Basic to Clinical. 2923-2951. DOI: 10.1007/978-1-4614-1997-6_113  0.67
2010 Abrajano JJ, Qureshi IA, Gokhan S, Molero AE, Zheng D, Bergman A, Mehler MF. Corepressor for element-1-silencing transcription factor preferentially mediates gene networks underlying neural stem cell fate decisions. Proceedings of the National Academy of Sciences of the United States of America. 107: 16685-90. PMID 20823235 DOI: 10.1073/Pnas.0906917107  0.727
2009 Molero AE, Gokhan S, Gonzalez S, Feig JL, Alexandre LC, Mehler MF. Impairment of developmental stem cell-mediated striatal neurogenesis and pluripotency genes in a knock-in model of Huntington's disease. Proceedings of the National Academy of Sciences of the United States of America. 106: 21900-5. PMID 19955426 DOI: 10.1073/Pnas.0912171106  0.773
2002 Yung SY, Gokhan S, Jurcsak J, Molero AE, Abrajano JJ, Mehler MF. Differential modulation of BMP signaling promotes the elaboration of cerebral cortical GABAergic neurons or oligodendrocytes from a common sonic hedgehog-responsive ventral forebrain progenitor species. Proceedings of the National Academy of Sciences of the United States of America. 99: 16273-8. PMID 12461181 DOI: 10.1073/Pnas.232586699  0.69
Low-probability matches (unlikely to be authored by this person)
2006 Pino-Ramirez G, Pineda-Manzano NC, Molero AE, Maestre GE. P3-010: Neuropsychological profiles of Alzheimer disease and vascular dementia. Findings from the Maracaibo aging study Alzheimers & Dementia. 2. DOI: 10.1016/J.Jalz.2006.05.1277  0.258
2001 Molero AE, Pino-Ramírez G, Maestre GE. Modulation by age and gender of risk for Alzheimer's disease and vascular dementia associated with the apolipoprotein E-ε4 allele in Latin Americans: Findings from the Maracaibo Aging Study Neuroscience Letters. 307: 5-8. PMID 11516561 DOI: 10.1016/S0304-3940(01)01911-5  0.248
2009 Chacón IJ, Molero AE, Pino-Ramírez G, Luchsinger JA, Lee JH, Maestre GE. Risk of dementia associated with elevated plasma homocysteine in a latin american population. International Journal of Alzheimer's Disease. 2009. PMID 20798752 DOI: 10.4061/2009/632489  0.235
2007 Molero AE, Pino-Ramírez G, Maestre GE. High prevalence of dementia in a Caribbean population. Neuroepidemiology. 29: 107-12. PMID 17940342 DOI: 10.1159/000109824  0.218
2002 Maestre GE, Pino-Ramírez G, Molero AE, Silva ER, Zambrano R, Falque L, Gamero MP, Sulbarán TA. The Maracaibo Aging Study: population and methodological issues. Neuroepidemiology. 21: 194-201. PMID 12065882 DOI: 10.1159/000059524  0.214
2006 Molero AE, Altimari CC, Duran DA, Garcia E, Pino-Ramirez G, Maestre GE. Total plasma homocysteine values among elderly subjects: findings from the Maracaibo Aging Study. Clinical Biochemistry. 39: 1007-15. PMID 16959233 DOI: 10.1016/J.Clinbiochem.2006.07.005  0.196
2006 Chacon IJ, Molero A, Pino G, Lee JH, Luchsinger JA, Maestre GE. P3-113: Plasma homocysteine and risk of dementia in a hispanic population Alzheimers & Dementia. 2. DOI: 10.1016/J.Jalz.2006.05.1381  0.184
2004 Pino-Ramírez G, Molero AE, Maestre GE. P1-101 The uses of reference value methodology for cutoff estimation in neuropsychological diagnostic tests for dementia Neurobiology of Aging. 25. DOI: 10.1016/S0197-4580(04)80415-9  0.179
2000 Pino-Ramirez G, Molero AE, Castellano MZ, Rodriguez MY, Maestre GE. Dementia care in Maracaibo, Venezuela Neurobiology of Aging. 21: 195. DOI: 10.1016/S0197-4580(00)82221-6  0.178
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