Roberto Zayas, Ph.D. - Publications
Affiliations: | 2005 | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
Neuroscience Biology, Cell BiologyYear | Citation | Score | |||
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2007 | Groshong JS, Spencer MJ, Bhattacharyya BJ, Kudryashova E, Vohra BP, Zayas R, Wollmann RL, Miller RJ, Gomez CM. Calpain activation impairs neuromuscular transmission in a mouse model of the slow-channel myasthenic syndrome. The Journal of Clinical Investigation. 117: 2903-12. PMID 17853947 DOI: 10.1172/Jci30383 | 0.638 | |||
2007 | Zayas R, Groshong JS, Gomez CM. Inositol-1,4,5-triphosphate receptors mediate activity-induced synaptic Ca2+ signals in muscle fibers and Ca2+ overload in slow-channel syndrome. Cell Calcium. 41: 343-52. PMID 16973214 DOI: 10.1016/J.Ceca.2006.07.007 | 0.552 | |||
2006 | Zayas R, Lasalde-Dominicci J, Gomez CM. Macroscopic properties of spontaneous mutations in slow-channel syndrome: correlation by domain and disease severity. Synapse (New York, N.Y.). 60: 441-9. PMID 16881075 DOI: 10.1002/Syn.20317 | 0.622 | |||
2006 | Vohra BP, Groshong JS, Zayas R, Wollmann RL, Gomez CM. Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model. Neurobiology of Disease. 23: 462-70. PMID 16815027 DOI: 10.1016/J.Nbd.2006.04.018 | 0.572 | |||
2006 | Navedo MF, Lasalde-Dominicci JA, Báez-Pagán CA, DÃaz-Pérez L, Rojas LV, Maselli RA, Staub J, Schott K, Zayas R, Gomez CM. Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociation. Molecular and Cellular Neurosciences. 32: 82-90. PMID 16624571 DOI: 10.1016/J.Mcn.2006.02.004 | 0.62 | |||
2002 | Gomez CM, Maselli RA, Groshong J, Zayas R, Wollmann RL, Cens T, Charnet P. Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 22: 6447-57. PMID 12151524 DOI: 10.1523/Jneurosci.22-15-06447.2002 | 0.637 | |||
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