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David Acton - Publications

Affiliations: 
2017- Molecular Neuroscience Laboratories Salk Institute for Biological Studies, La Jolla, CA, United States 
Area:
Glia, gliotransmission, spinal cord, electrophysiology, locomotion
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7 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2023 Ren X, Liu S, Virlogeux A, Kang SJ, Brusch J, Liu Y, Dymecki SM, Han S, Goulding M, Acton D. Identification of an essential spinoparabrachial pathway for mechanical itch. Neuron. PMID 37023756 DOI: 10.1016/j.neuron.2023.03.013  0.734
2019 Acton D, Ren X, Di Costanzo S, Dalet A, Bourane S, Bertocchi I, Eva C, Goulding M. Spinal Neuropeptide Y1 Receptor-Expressing Neurons Form an Essential Excitatory Pathway for Mechanical Itch. Cell Reports. 28: 625-639.e6. PMID 31315043 DOI: 10.1016/J.Celrep.2019.06.033  0.642
2018 Acton D, Broadhead MJ, Miles GB. Modulation of spinal motor networks by astrocyte-derived adenosine is dependent on D1-like dopamine receptor signalling. Journal of Neurophysiology. PMID 29790837 DOI: 10.1152/Jn.00783.2017  0.692
2017 Koch SC, Acton D, Goulding M. Spinal Circuits for Touch, Pain, and Itch. Annual Review of Physiology. PMID 28961064 DOI: 10.1146/Annurev-Physiol-022516-034303  0.702
2017 Acton D, Miles GB. Gliotransmission and adenosinergic modulation: insights from mammalian spinal motor networks. Journal of Neurophysiology. jn.00230.2017. PMID 28954893 DOI: 10.1152/Jn.00230.2017  0.746
2017 Acton D, Miles GB. Differential regulation of NMDA receptors by D-serine and glycine in mammalian spinal locomotor networks. Journal of Neurophysiology. jn.00810.2016. PMID 28202572 DOI: 10.1152/Jn.00810.2016  0.685
2015 Acton D, Miles GB. Stimulation of Glia Reveals Modulation of Mammalian Spinal Motor Networks by Adenosine. Plos One. 10: e0134488. PMID 26252389 DOI: 10.1371/Journal.Pone.0134488  0.741
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