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Ronan Vinicius da Silva - Publications

Affiliations: 
IRCM 
Area:
Pain system development, Spinothalamic tract
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5 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2018 da Silva RV, Johannssen HC, Wyss MT, Roome RB, Bourojeni FB, Stifani N, Marsh APL, Ryan MM, Lockhart PJ, Leventer RJ, Richards LJ, Rosenblatt B, Srour M, Weber B, Zeilhofer HU, et al. DCC Is Required for the Development of Nociceptive Topognosis in Mice and Humans. Cell Reports. 22: 1105-1114. PMID 29386099 DOI: 10.1016/J.Celrep.2018.01.004  0.346
2017 Peng J, Ferent J, Li Q, Liu M, da Silva RV, Zeilhofer HU, Kania A, Zhang Y, Charron F. Loss of Dcc in the spinal cord is sufficient to cause a deficit in lateralized motor control and the switch to a hopping gait. Developmental Dynamics : An Official Publication of the American Association of Anatomists. PMID 28691197 DOI: 10.1002/Dvdy.24549  0.568
2015 Szabo N, Da Silva RV, Sotocinal SG, Mogil JS, Kania A. A role for the transcription factor lmx1b in pain modality discrimination. International Journal of Developmental Neuroscience : the Official Journal of the International Society For Developmental Neuroscience. 47: 115-6. PMID 26531633 DOI: 10.1016/J.Ijdevneu.2015.04.312  0.583
2015 Szabo NE, da Silva RV, Sotocinal SG, Zeilhofer HU, Mogil JS, Kania A. Hoxb8 intersection defines a role for Lmx1b in excitatory dorsal horn neuron development, spinofugal connectivity, and nociception. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 5233-46. PMID 25834049 DOI: 10.1523/JNEUROSCI.4690-14.2015  0.682
2014 Laumonnerie C, Da Silva RV, Kania A, Wilson SI. Netrin 1 and Dcc signalling are required for confinement of central axons within the central nervous system. Development (Cambridge, England). 141: 594-603. PMID 24449837 DOI: 10.1242/dev.099606  0.495
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