Year |
Citation |
Score |
2012 |
Guldal CG, Ahmad A, Korshunov A, Squatrito M, Awan A, Mainwaring LA, Bhatia B, Parathath SR, Nahle Z, Pfister S, Kenney AM. An essential role for p38 MAPK in cerebellar granule neuron precursor proliferation. Acta Neuropathologica. 123: 573-86. PMID 22302101 DOI: 10.1007/S00401-012-0946-Z |
0.672 |
|
2011 |
Mainwaring LA, Kenney AM. Divergent functions for eIF4E and S6 kinase by sonic hedgehog mitogenic signaling in the developing cerebellum. Oncogene. 30: 1784-97. PMID 21339731 DOI: 10.1038/Onc.2010.564 |
0.686 |
|
2010 |
Mainwaring LA, Bhatia B, Kenney AM. Myc on my mind: a transcription factor family's essential role in brain development. Oncotarget. 1: 86-8. PMID 21297222 DOI: 10.18632/Oncotarget.100611 |
0.639 |
|
2010 |
Parathath SR, Mainwaring LA, Fernandez-L A, Guldal CG, Nahlé Z, Kenney AM. β-arrestin-1 links mitogenic sonic hedgehog signaling to the cell cycle exit machinery in neural precursors Cell Cycle. 9: 4013-4024. PMID 20935513 DOI: 10.4161/Cc.9.19.13325 |
0.683 |
|
2010 |
Cemile GG, Mainwaring L, Parathath S, Kenney AM. Abstract LB-13: The role of p38/MAPK14 in Sonic Hedgehog-driven CGNP proliferation Cancer Research. 70. DOI: 10.1158/1538-7445.Am10-Lb-13 |
0.545 |
|
2008 |
Parathath SR, Mainwaring LA, Fernandez-L A, Campbell DO, Kenney AM. Insulin receptor substrate 1 is an effector of sonic hedgehog mitogenic signaling in cerebellar neural precursors Development. 135: 3291-3300. PMID 18755774 DOI: 10.1242/Dev.022871 |
0.679 |
|
2008 |
Becher OJ, Hambardzumyan D, Fomchenko EI, Momota H, Mainwaring L, Bleau AM, Katz AM, Edgar M, Kenney AM, Cordon-Cardo C, Blasberg RG, Holland EC. Gli activity correlates with tumor grade in platelet-derived growth factor-induced gliomas. Cancer Research. 68: 2241-9. PMID 18381430 DOI: 10.1158/0008-5472.Can-07-6350 |
0.62 |
|
2004 |
Sloand EM, Ramkissoon S, Mainwaring L, Barrett J, Young NS. Granulocyte Colony Stimulating Factor Preferentially Causes Proliferation of Pre-Existing Monosomy 7 Cells in Aplastic Anemia and Myelodysplastic Syndrome Because of Abnormal Truncated GCSF-Receptors on These Cells. Blood. 104: 458-458. DOI: 10.1182/BLOOD.V104.11.458.458 |
0.325 |
|
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