Hartmut Cuny - Publications

Affiliations: 
2009-2015 RMIT University, Melbourne, Victoria, Australia 

10/17 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any innacuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2017 Cuny H, Yu R, Tae HS, Kompella SN, Adams DJ. α-Conotoxins active at α3-containing nicotinic acetylcholine receptors and their molecular determinants for selective inhibition. British Journal of Pharmacology. PMID 28477355 DOI: 10.1111/bph.13852  0.8
2016 Cuny H, Kompella SN, Tae HS, Yu R, Adams DJ. Key Structural Determinants in the Agonist Binding Loops of Human β2 and β4 Nicotinic Acetylcholine Receptor Subunits Contribute to α3β4 Subtype Selectivity of α-Conotoxins. The Journal of Biological Chemistry. PMID 27646000 DOI: 10.1074/jbc.M116.730804  0.8
2015 Kompella SN, Cuny H, Hung A, Adams DJ. Molecular Basis for Differential Sensitivity of α-Conotoxin RegIIA at Rat and Human Neuronal Nicotinic Acetylcholine Receptors. Molecular Pharmacology. 88: 993-1001. PMID 26438212 DOI: 10.1124/mol.115.100503  0.8
2015 Huynh TG, Cuny H, Slesinger PA, Adams DJ. Novel mechanism of voltage-gated N-type (Cav2.2) calcium channel inhibition revealed through α-conotoxin Vc1.1 activation of the GABA(B) receptor. Molecular Pharmacology. 87: 240-50. PMID 25425625 DOI: 10.1124/mol.114.096156  0.8
2014 Grimm C, Holdt LM, Chen CC, Hassan S, Müller C, Jörs S, Cuny H, Kissing S, Schröder B, Butz E, Northoff B, Castonguay J, Luber CA, Moser M, Spahn S, et al. High susceptibility to fatty liver disease in two-pore channel 2-deficient mice. Nature Communications. 5: 4699. PMID 25144390 DOI: 10.1038/ncomms5699  0.8
2014 Berecki G, McArthur JR, Cuny H, Clark RJ, Adams DJ. Differential Cav2.1 and Cav2.3 channel inhibition by baclofen and α-conotoxin Vc1.1 via GABAB receptor activation. The Journal of General Physiology. 143: 465-79. PMID 24688019 DOI: 10.1085/jgp.201311104  0.8
2013 Grishin AA, Cuny H, Hung A, Clark RJ, Brust A, Akondi K, Alewood PF, Craik DJ, Adams DJ. Identifying key amino acid residues that affect α-conotoxin AuIB inhibition of α3β4 nicotinic acetylcholine receptors. The Journal of Biological Chemistry. 288: 34428-42. PMID 24100032 DOI: 10.1074/jbc.M113.512582  0.8
2013 Inserra MC, Kompella SN, Vetter I, Brust A, Daly NL, Cuny H, Craik DJ, Alewood PF, Adams DJ, Lewis RJ. Isolation and characterization of α-conotoxin LsIA with potent activity at nicotinic acetylcholine receptors. Biochemical Pharmacology. 86: 791-9. PMID 23924607 DOI: 10.1016/j.bcp.2013.07.016  0.8
2013 Yasuda T, Cuny H, Adams DJ. Kv3.1 channels stimulate adult neural precursor cell proliferation and neuronal differentiation. The Journal of Physiology. 591: 2579-91. PMID 23478135 DOI: 10.1113/jphysiol.2012.249151  0.8
2012 Cuny H, de Faoite A, Huynh TG, Yasuda T, Berecki G, Adams DJ. γ-Aminobutyric acid type B (GABAB) receptor expression is needed for inhibition of N-type (Cav2.2) calcium channels by analgesic α-conotoxins. The Journal of Biological Chemistry. 287: 23948-57. PMID 22613715 DOI: 10.1074/jbc.M112.342998  0.8
Low-probability matches
2020 Cuny H, Rapadas M, Gereis J, Martin EMMA, Kirk RB, Shi H, Dunwoodie SL. NAD deficiency due to environmental factors or gene-environment interactions causes congenital malformations and miscarriage in mice. Proceedings of the National Academy of Sciences of the United States of America. PMID 32015132 DOI: 10.1073/pnas.1916588117  0.01
2019 Szot JO, Campagnolo C, Cao Y, Iyer KR, Cuny H, Drysdale T, Flores-Daboub JA, Bi W, Westerfield L, Liu P, Leung TN, Choy KW, Chapman G, Xiao R, Siu VM, et al. Bi-Allelic Mutations in NADSYN1 Cause Multiple Organ Defects and Expand the Genotypic Spectrum of Congenital NAD Deficiency Disorders. American Journal of Human Genetics. PMID 31883644 DOI: 10.1016/j.ajhg.2019.12.006  0.01
2018 Alankarage D, Ip E, Szot JO, Munro J, Blue GM, Harrison K, Cuny H, Enriquez A, Troup M, Humphreys DT, Wilson M, Harvey RP, Sholler GF, Graham RM, Ho JWK, et al. Identification of clinically actionable variants from genome sequencing of families with congenital heart disease. Genetics in Medicine : Official Journal of the American College of Medical Genetics. PMID 30293987 DOI: 10.1038/s41436-018-0296-x  0.01
2018 Szot JO, Cuny H, Blue GM, Humphreys DT, Ip E, Harrison K, Sholler GF, Giannoulatou E, Leo P, Duncan EL, Sparrow DB, Ho JWK, Graham RM, Pachter N, Chapman G, et al. A Screening Approach to Identify Clinically Actionable Variants Causing Congenital Heart Disease in Exome Data. Circulation. Genomic and Precision Medicine. 11: e001978. PMID 29555671 DOI: 10.1161/CIRCGEN.117.001978  0.01
2009 Griessmeier K, Cuny H, Rötzer K, Griesbeck O, Harz H, Biel M, Wahl-Schott C. Calmodulin is a functional regulator of Cav1.4 L-type Ca2+ channels. The Journal of Biological Chemistry. 284: 29809-16. PMID 19717559 DOI: 10.1074/jbc.M109.048082  0.01
2009 Zong X, Schieder M, Cuny H, Fenske S, Gruner C, Rötzer K, Griesbeck O, Harz H, Biel M, Wahl-Schott C. The two-pore channel TPCN2 mediates NAADP-dependent Ca(2+)-release from lysosomal stores. Pflugers Archiv : European Journal of Physiology. 458: 891-9. PMID 19557428 DOI: 10.1007/s00424-009-0690-y  0.01
2006 Wahl-Schott C, Baumann L, Cuny H, Eckert C, Griessmeier K, Biel M. Switching off calcium-dependent inactivation in L-type calcium channels by an autoinhibitory domain. Proceedings of the National Academy of Sciences of the United States of America. 103: 15657-62. PMID 17028172 DOI: 10.1073/pnas.0604621103  0.01
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