| Year |
Citation |
Score |
| 2017 |
Goiran T, Duplan E, Chami M, Bourgeois A, El Manaa W, Rouland L, Dunys J, Lauritzen I, You H, Stambolic V, Biféri MG, Barkats M, Pimplikar SW, Sergeant N, Colin M, et al. β-Amyloid Precursor Protein Intracellular Domain Controls Mitochondrial Function by Modulating Phosphatase and Tensin Homolog-Induced Kinase 1 Transcription in Cells and in Alzheimer Mice Models. Biological Psychiatry. PMID 28587718 DOI: 10.1016/j.biopsych.2017.04.011 |
0.454 |
|
| 2017 |
Pimplikar SW. Multi-omics and Alzheimer's disease: a slower but surer path to an efficacious therapy? American Journal of Physiology. Cell Physiology. 313: C1-C2. PMID 28515086 DOI: 10.1152/ajpcell.00109.2017 |
0.324 |
|
| 2016 |
Ghosal K, Fan Q, Dawson HN, Pimplikar SW. Tau Protein Mediates APP Intracellular Domain (AICD)-Induced Alzheimer's-Like Pathological Features in Mice. Plos One. 11: e0159435. PMID 27459671 DOI: 10.1371/journal.pone.0159435 |
0.644 |
|
| 2015 |
Margevicius DR, Bastian C, Fan Q, Davis RJ, Pimplikar SW. JNK-interacting protein 1 mediates Alzheimer's-like pathological features in AICD-transgenic mice. Neurobiology of Aging. 36: 2370-9. PMID 26022769 DOI: 10.1016/J.Neurobiolaging.2015.04.013 |
0.653 |
|
| 2015 |
Jay TR, Miller CM, Cheng PJ, Graham LC, Bemiller S, Broihier ML, Xu G, Margevicius D, Karlo JC, Sousa GL, Cotleur AC, Butovsky O, Bekris L, Staugaitis SM, Leverenz JB, ... Pimplikar SW, et al. TREM2 deficiency eliminates TREM2+ inflammatory macrophages and ameliorates pathology in Alzheimer's disease mouse models. The Journal of Experimental Medicine. 212: 287-95. PMID 25732305 DOI: 10.1084/Jem.20142322 |
0.588 |
|
| 2014 |
Pimplikar SW. Neuroinflammation in Alzheimer's disease: from pathogenesis to a therapeutic target. Journal of Clinical Immunology. 34: S64-9. PMID 24711006 DOI: 10.1007/s10875-014-0032-5 |
0.549 |
|
| 2013 |
Ghosal K, Stathopoulos A, Thomas D, Phenis D, Vitek MP, Pimplikar SW. The apolipoprotein-E-mimetic COG112 protects amyloid precursor protein intracellular domain-overexpressing animals from Alzheimer's disease-like pathological features. Neuro-Degenerative Diseases. 12: 51-8. PMID 22965147 DOI: 10.1159/000341299 |
0.642 |
|
| 2012 |
Pimplikar SW. Clinical and biomarker changes in Alzheimer's disease. The New England Journal of Medicine. 367: 2050-1; author reply. PMID 23171105 DOI: 10.1056/NEJMc1211767#SA2 |
0.353 |
|
| 2012 |
Song P, Pimplikar SW. Knockdown of amyloid precursor protein in zebrafish causes defects in motor axon outgrowth Plos One. 7. PMID 22545081 DOI: 10.1371/journal.pone.0034209 |
0.441 |
|
| 2011 |
Pimplikar SW, Ghosal K. Amyloid precursor protein: more than just neurodegeneration. Stem Cell Research & Therapy. 2: 39. PMID 22000643 DOI: 10.1186/scrt80 |
0.502 |
|
| 2011 |
Pimplikar SW, Suryanarayana A. Detection of APP intracellular domain in brain tissue. Methods in Molecular Biology (Clifton, N.J.). 670: 85-91. PMID 20967585 DOI: 10.1007/978-1-60761-744-0_7 |
0.467 |
|
| 2011 |
Ghosal K, Pimplikar SW. Aging and excitotoxic stress exacerbate neural circuit reorganization in amyloid precursor protein intracellular domain transgenic mice. Neurobiology of Aging. 32: 2320.e1-9. PMID 20493588 DOI: 10.1016/j.neurobiolaging.2010.04.020 |
0.544 |
|
| 2011 |
Briand S, Facchinetti P, Clamagirand C, Madeira A, Pommet JM, Pimplikar SW, Allinquant B. PAT1 induces cell death signal and SET mislocalization into the cytoplasm by increasing APP/APLP2 at the cell surface. Neurobiology of Aging. 32: 1099-113. PMID 19570594 DOI: 10.1016/j.neurobiolaging.2009.05.021 |
0.388 |
|
| 2011 |
Pimplikar S, Ghosal K, Fan Q. Amyloid-independent mechanisms of Alzheiemer's disease: The role of APP intracellular domain Alzheimers & Dementia. 7. DOI: 10.1016/J.Jalz.2011.05.1206 |
0.441 |
|
| 2010 |
Pimplikar SW, Nixon RA, Robakis NK, Shen J, Tsai LH. Amyloid-independent mechanisms in Alzheimer's disease pathogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 14946-54. PMID 21068297 DOI: 10.1523/Jneurosci.4305-10.2010 |
0.644 |
|
| 2010 |
Lazarov O, Mattson MP, Peterson DA, Pimplikar SW, van Praag H. When neurogenesis encounters aging and disease. Trends in Neurosciences. 33: 569-79. PMID 20961627 DOI: 10.1016/J.Tins.2010.09.003 |
0.426 |
|
| 2010 |
Ghosal K, Stathopoulos A, Pimplikar SW. APP intracellular domain impairs adult neurogenesis in transgenic mice by inducing neuroinflammation. Plos One. 5: e11866. PMID 20689579 DOI: 10.1371/journal.pone.0011866 |
0.679 |
|
| 2009 |
Ghosal K, Vogt DL, Liang M, Shen Y, Lamb BT, Pimplikar SW. Alzheimer's disease-like pathological features in transgenic mice expressing the APP intracellular domain. Proceedings of the National Academy of Sciences of the United States of America. 106: 18367-72. PMID 19837693 DOI: 10.1073/Pnas.0907652106 |
0.355 |
|
| 2009 |
Joshi P, Liang JO, DiMonte K, Sullivan J, Pimplikar SW. Amyloid precursor protein is required for convergent-extension movements during Zebrafish development. Developmental Biology. 335: 1-11. PMID 19664615 DOI: 10.1016/j.ydbio.2009.07.041 |
0.533 |
|
| 2009 |
Pimplikar SW. Reassessing the amyloid cascade hypothesis of Alzheimer's disease. The International Journal of Biochemistry & Cell Biology. 41: 1261-8. PMID 19124085 DOI: 10.1016/j.biocel.2008.12.015 |
0.663 |
|
| 2008 |
Varvel NH, Bhaskar K, Patil AR, Pimplikar SW, Herrup K, Lamb BT. Abeta oligomers induce neuronal cell cycle events in Alzheimer's disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 10786-93. PMID 18945886 DOI: 10.1523/Jneurosci.2441-08.2008 |
0.492 |
|
| 2005 |
Ryan KA, Pimplikar SW. Activation of GSK-3 and phosphorylation of CRMP2 in transgenic mice expressing APP intracellular domain Journal of Cell Biology. 171: 327-335. PMID 16230462 DOI: 10.1083/jcb.200505078 |
0.409 |
|
| 2002 |
Pimplikar SW. AbetaPP, apoptosis and Alzheimer's disease. Journal of Alzheimer's Disease : Jad. 4: 39-40. PMID 12214017 |
0.426 |
|
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