Syed Rydwan Ali - Publications

Affiliations: 
2011-2016 Pharmacology University of Texas Medical Branch, Galveston, TX 

18 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2021 Singh AK, Dvorak NM, Tapia CM, Mosebarger A, Ali SR, Bullock Z, Chen H, Zhou J, Laezza F. Differential Modulation of the Voltage-Gated Na Channel 1.6 by Peptides Derived From Fibroblast Growth Factor 14. Frontiers in Molecular Biosciences. 8: 742903. PMID 34557523 DOI: 10.3389/fmolb.2021.742903  0.789
2021 Zhang Y, Ali SR, Nabbout R, Barcia G, Kaczmarek LK. A KCNC1 mutation in Epilepsy of Infancy with Focal Migrating Seizures produces functional channels that fail to be regulated by phosphorylation. Journal of Neurophysiology. PMID 34232791 DOI: 10.1152/jn.00257.2021  0.673
2020 Singh AK, Wadsworth PA, Tapia CM, Aceto G, Ali SR, Chen H, D'Ascenzo M, Zhou J, Laezza F. Mapping of the FGF14:Nav1.6 complex interface reveals FLPK as a functionally active peptide modulating excitability. Physiological Reports. 8: e14505. PMID 32671946 DOI: 10.14814/Phy2.14505  0.777
2020 Ali SR, Malone TJ, Zhang Y, Prechova M, Kaczmarek LK. Phactr1 regulates Slack (KCNT1) channels via protein phosphatase 1 (PP1). Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 34: 1591-1601. PMID 31914597 DOI: 10.1096/Fj.201902366R  0.747
2019 Quraishi IH, Stern S, Mangan KP, Zhang Y, Ali SR, Mercier MR, Marchetto MC, McLachlan MJ, Jones EM, Gage FH, Kaczmarek LK. An epilepsy-associated KCNT1 mutation enhances excitability of human iPSC-derived neurons by increasing Slack K currents. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. PMID 31350261 DOI: 10.1523/Jneurosci.1628-18.2019  0.763
2018 Liu Z, Wadsworth P, Singh AK, Chen H, Wang P, Folorunso O, Scaduto P, Ali SR, Laezza F, Zhou J. Identification of peptidomimetics as novel chemical probes modulating fibroblast growth factor 14 (FGF14) and voltage-gated sodium channel 1.6 (Nav1.6) protein-protein interactions. Bioorganic & Medicinal Chemistry Letters. PMID 30587448 DOI: 10.1016/J.Bmcl.2018.12.031  0.816
2018 Ali SR, Liu Z, Nenov MN, Folorunso O, Singh AK, Scala F, Chen H, James TF, Alshammari M, Panova-Elektronova NI, White MA, Zhou J, Laezza F. Functional modulation of voltage-gated sodium channels by a FGF14-based peptidomimetic. Acs Chemical Neuroscience. PMID 29359916 DOI: 10.1021/Acschemneuro.7B00399  0.811
2018 Ali SR, Kaczmarek LK. The Functional Modulation of Sodium-Activated Potassium (Slack) Channels by Phosphatase and Actin Regulator 1 (Phactr1) Biophysical Journal. 114: 295a-296a. DOI: 10.1016/J.Bpj.2017.11.1687  0.666
2017 Wong J, Ali SR, Wadsworth P, Singh AK, Liu Z, Chen H, Zhou J, Laezza F. Virtual Screening in Search of Allosteric Modulators of Nav1.1 Channels Biophysical Journal. 112: 242a. DOI: 10.1016/J.Bpj.2016.11.1325  0.787
2016 Ali SR, Singh AK, Laezza F. Identification of Amino Acid Residues in the Fibroblast Growth Factor 14 (FGF14) Required For Structure-Function interactions with the Voltage-Gated Sodium Channel Nav1.6. The Journal of Biological Chemistry. PMID 26994141 DOI: 10.1074/Jbc.M115.703868  0.816
2016 Singh AK, Ali SR, Laezza F. Defining the Protein:Protein Interaction Interface of FGF14:Nav1.6 Complex Biophysical Journal. 110: 438a. DOI: 10.1016/J.Bpj.2015.11.2362  0.78
2015 Shavkunov AS, Ali SR, Panova-Elektronova NI, Laezza F. Split-luciferase complementation assay to detect channel-protein interactions in live cells. Methods in Molecular Biology (Clifton, N.J.). 1278: 497-514. PMID 25859972 DOI: 10.1007/978-1-4939-2425-7_33  0.793
2015 Wildburger NC, Ali SR, Hsu WC, Shavkunov AS, Nenov MN, Lichti CF, LeDuc RD, Mostovenko E, Panova-Elektronova NI, Emmett MR, Nilsson CL, Laezza F. Quantitative proteomics reveals protein-protein interactions with fibroblast growth factor 12 as a component of the voltage-gated sodium channel 1.2 (nav1.2) macromolecular complex in Mammalian brain. Molecular & Cellular Proteomics : McP. 14: 1288-300. PMID 25724910 DOI: 10.1074/Mcp.M114.040055  0.73
2015 Ali SR, Liu Z, Nenov MN, Panova-Elektro NI, Zhou J, Stoilova-McPhie S, Laezza F. Targeting Protein:Protein Interaction Sites for Drug Development against Voltage-Gated Sodium Channels Biophysical Journal. 108: 574a. DOI: 10.1016/J.Bpj.2014.11.3140  0.833
2014 Ali S, Shavkunov A, Panova N, Stoilova-McPhie S, Laezza F. Modulation of the FGF14:FGF14 homodimer interaction through short peptide fragments. Cns & Neurological Disorders Drug Targets. 13: 1559-70. PMID 25426956 DOI: 10.2174/1871527313666141126103309  0.81
2014 Ali SR, Panova N, Stoilova-McPhie S, Laezza F. Protein-Protein Interactions Based Drug Discovery Against the Voltage-Gated Sodium Channel Biophysical Journal. 106: 326a. DOI: 10.1016/J.Bpj.2013.11.1875  0.822
2013 Stoilova-McPhie S, Ali S, Laezza F. Protein-Protein Interactions as New Targets for Ion Channel Drug Discovery. Austin Journal of Pharmacology and Therapeutics. 1. PMID 25485305  0.739
2011 Liu Y, Lo YC, Qian L, Crews FT, Wilson B, Chen HL, Wu HM, Chen SH, Wei K, Lu RB, Ali S, Hong JS. Verapamil protects dopaminergic neuron damage through a novel anti-inflammatory mechanism by inhibition of microglial activation. Neuropharmacology. 60: 373-80. PMID 20950631 DOI: 10.1016/J.Neuropharm.2010.10.002  0.344
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